Vitamin K is a substance necessary for the liver to synthesize coagulation factors II, VII, IX and X. Vitamin K deficiency can cause synthesis disorders of these coagulation factors, and clinically, bleeding tendency and prolonged prothrombin time can be seen. Natural vitamin K includes K1 and K2, which are fat-soluble and their absorption depends on the normal secretion of bile. Artificially synthesized vitamin K includes K3 and K4, the former is sodium bisulfite menadione, and the latter is acetyl menadione, which are water-soluble and do not require the presence of bile for their absorption.

This product is the earliest sulfonamide antibiotic used for intestinal infections.

The antibacterial spectrum is similar to that of rifampicin. It has good antibacterial activity against tuberculosis and leprosy. When the dosage is 1/3 of that of rifampicin, similar or higher efficacy can be achieved. It has a good effect on Staphylococcus aureus and has certain antibacterial activity against some Escherichia coli. In addition, it also has an inhibitory effect on Chlamydia trachomatis.

It is a Class Ib antiarrhythmic drug that can inhibit sodium influx into myocardial cells, reduce the speed of phase 0 depolarization of action potential, and shorten the effective refractory period of Purkinje fibers. In patients with normal cardiac conduction system, it has little effect on the generation and conduction of cardiac impulses, does not prolong the duration of ventricular depolarization and repolarization, and is suitable for ventricular arrhythmias with prolonged QT interval. It has antiarrhythmic, anticonvulsant and local anesthetic effects. It has a small inhibitory effect on the myocardium.

It acts on the lower brain stem of the central nervous system, inhibits multi-synaptic reflexes, blocks nerve conduction from abnormally excited muscles, and produces muscle relaxation. It also acts on the higher centers of the cerebral cortex and has a weak sedative effect.