1. It can obviously inhibit the synthesis of thromboxane A2 (TXA2), counteract the platelet aggregation caused by arachidonic acid (AA), and has a stronger and faster antiplatelet aggregation effect than aspirin (ASP). 2. It can enhance the fibrinolytic activity and promote the dissolution of thrombus. 3. It can reduce the specific viscosity of whole blood and plasma, and accelerate the electrophoresis of red blood cells. 4. It can relax the vascular smooth muscle, dilate the artery, and increase the blood flow of the coronary artery and cerebral artery. 5. It can improve the ability to resist hypoxia. 6. It can strongly scavenge free radicals. 7. It can inhibit the inflammatory exudation of capillaries.

It acts directly on the endogenous fibrinolytic system, catalyzing the cleavage of plasminogen into plasmin, degrading fibrin clots, fibrinogen in the blood circulation, coagulation factor V and coagulation factor VIII, etc.; it increases the activity of vascular ADPase, inhibits ADP-induced platelet aggregation; it shows that the thrombolytic effect is significantly related to the drug dosage and the time window of administration; it may increase the risk of severe bleeding.

It acts directly on the endogenous fibrinolytic system, catalyzing the cleavage of plasminogen into plasmin, degrading fibrin clots, fibrinogen in the blood circulation, coagulation factor V and coagulation factor VIII, etc.; it increases the activity of vascular ADPase, inhibits ADP-induced platelet aggregation; it shows that the thrombolytic effect is significantly related to the drug dosage and the time window of administration; it may increase the risk of severe bleeding.

It is a protein kinase activator and a derivative of nucleotides. It is an important substance with physiological activity that is widely present in the human body. It is produced by adenosine triphosphate under the catalysis of adenylate cyclase and can regulate various functional activities of cells. As the second messenger of hormones, it plays a role in regulating physiological functions and material metabolism in cells, can change the function of cell membranes, promote calcium ions in reticulum to enter muscle fibers, thereby enhancing myocardial contraction, and can promote the activity of respiratory chain oxidases, improve myocardial hypoxia, relieve symptoms of coronary heart disease and improve electrocardiograms. In addition, it plays an important role in regulating sugar, fat metabolism, nucleic acid, and protein synthesis.

Tetracycline antibiotics. This product is a broad-spectrum antibacterial agent with bactericidal effects at high concentrations. Rickettsia, Mycoplasma, Chlamydia, atypical mycobacteria, and spirochetes are also sensitive to this product. This product is more effective against Gram-positive bacteria than Gram-negative bacteria, but Enterococcus is resistant to it. Others such as Actinomycetes, Bacillus anthracis, Listeria monocytogenes, Clostridium, Nocardia, Vibrio, Brucella, Campylobacter, and Yersinia are sensitive to this product. This product has certain antibacterial activity against Neisseria gonorrhoeae, but penicillin-resistant Neisseria gonorrhoeae is also resistant to doxycycline. Due to the widespread use of tetracyclines over the years, common clinical pathogens have become seriously resistant to this product, including Gram-positive bacteria such as Staphylococcus and most Gram-negative bacilli. This product is different from tetracycline

In vitro tests have destructive or inhibitory effects on the morphology or proliferation of cell lines such as liver cancer, esophageal cancer, laryngeal cancer, lung cancer, and cervical cancer; in vivo tests have a certain inhibitory effect on Ehrlich ascites carcinoma, sarcoma-180 and solid liver cancer in mice, and can increase the respiratory control rate and lysosomal enzyme activity of mitochondria in ascites liver cancer H22 mice, interfere with cancer cell division, block it in the M phase, and affect its cycle speed; destroy the cancer cell skeleton (microfilaments, microtubules), affect the ultrastructure of cancer cells, and cause damage to mitochondria, microvilli and plasma membranes; can inhibit cancer cell DNA synthesis, but has no inhibitory effect on normal bone marrow cells, and can increase white blood cells.