This product is a cell metabolite existing in the human body, which can regulate the activity of multiple enzyme systems in glucose metabolism. It is reported that exogenous sodium fructose diphosphate can increase the concentration of adenosine triphosphate and creatine phosphate in cells by activating the activity of phosphofructokinase and pyruvate kinase, promote the influx of potassium ions, and is beneficial to the energy metabolism and glucose utilization of cells under ischemic and hypoxic conditions, thereby reducing damage to ischemic myocardium.

Calcium channel blockers inhibit smooth muscle contraction to relieve vasospasm, selectively act on cerebral vascular smooth muscle, dilate cerebral blood vessels, increase cerebral blood flow, and reduce ischemic brain damage; they have the effects of protecting and promoting memory and promoting intellectual recovery.

This product is a dihydropyridine calcium channel blocker. This product inhibits the transmembrane calcium ion influx of vascular smooth muscle and myocardium, but its vascular action is mainly vascular, so its vascular selectivity is relatively strong. This product causes the expansion of systemic blood vessels such as coronary arteries and renal arterioles, producing a hypotensive effect.

Dihydropyridine calcium antagonists selectively inhibit the transmembrane transport of calcium ions into myocardial cells and smooth muscle cells, dilate coronary arteries and peripheral blood vessels, reduce myocardial oxygen consumption, reduce cardiac load, and do not affect plasma calcium ion concentration. They can antagonize coronary artery spasm, inhibit myocardial contraction, reduce myocardial metabolism, dilate peripheral resistance vessels, lower blood pressure, and reduce cardiac afterload, while having no significant inhibitory effect on electrophysiological function.

1. Reserpine is an adrenergic neuron blocking antihypertensive drug. 2. It achieves antihypertensive, heart rate slowing and central nervous system inhibition effects by depleting adrenaline in peripheral sympathetic nerve endings, catecholamines and 5-hydroxytryptamine in the heart, brain and other tissues. 3. The antihypertensive effect is mainly achieved by reducing cardiac output and peripheral resistance and partially inhibiting cardiovascular reflexes. 4. The effect of slowing heart rate is not obvious in patients with normal heart rate, but it is obvious in patients with sinus tachycardia. 5. It acts on the hypothalamus to produce a sedative effect, which can relieve anxiety, tension and headaches in hypertensive patients.