This product is a glucocorticoid with anti-inflammatory, immunosuppressive, antitoxin and anti-shock effects. (1) Anti-inflammatory effect: It has an effect on inflammation caused by various causes except viruses. Glucocorticoids reduce and prevent tissue response to inflammation, thereby reducing the symptoms of inflammation. They can also inhibit the repair of tissues in the later stages of inflammation and reduce sequelae. (2) Immunosuppressive effect: It prevents or inhibits cell-mediated immune responses, delayed allergic reactions, and reduces the expansion of primary immune responses. (3) Anti-toxic and anti-shock effects: Glucocorticoids can improve the body's tolerance, reduce cell damage, and play a role in protecting the body. It also has the effects of dilating blood vessels, enhancing myocardial contractility, and improving microcirculation.

This product is a broad-spectrum semi-synthetic penicillin with extremely low toxicity. The antibacterial spectrum is similar to that of penicillin, and it has a strong antibacterial effect on grass-green streptococci, hemolytic streptococci, and enterococci. Diphtheria bacillus, anthrax bacillus, Gram-positive anaerobic cocci and bacilli are sensitive to this product; meningococci, pertussis bacilli, and Brucella are also very sensitive. Escherichia coli, Salmonella, Shigella dysenteriae, Proteus, influenza bacilli, pneumococci, gonococci, etc., all have varying degrees of drug resistance because some strains produce beta-lactamase. Among them, the resistance rate of Escherichia coli is as high as 50-60%. Enterobacter bacteria, Pseudomonas aeruginosa, and anaerobic Gram-negative bacilli are resistant to this product. It is mainly used to treat infectious diseases caused by sensitive Staphylococcus aureus, hemolytic streptococci, gonococci, meningococci, diphtheria, pertussis, influenza bacilli, Salmonella, Shigella, etc.; such as respiratory tract infections (pneumonia, acute and chronic bronchitis, whooping cough, etc.), gastroenteritis and digestive tract infections (liver and gallbladder infections, acute and chronic gastroenteritis, bacillary dysentery, typhoid and paratyphoid, etc.), urinary tract infections (gonorrhea, urethritis, cystitis, etc.), soft tissue infections and sepsis, meningitis, endocarditis, etc.

It can inhibit the synthesis and release of prostaglandins in the hypothalamus, restore the normal responsiveness of the sensory neurons in the temperature regulation center, and have an antipyretic effect; at the same time, it also has an analgesic effect by inhibiting the synthesis of prostaglandins, etc. It can also inhibit the synthesis and release of prostaglandins in the local tissue of inflammation, stabilize lysosomal enzymes, and affect the phagocytic function of phagocytes to play an anti-inflammatory role.

It is a peripheral analgesic. Its mechanism of action is to inhibit the synthesis and release of prostaglandins (PGE1) in the hypothalamic temperature regulation center, thereby causing peripheral vasodilation and sweating to achieve an antipyretic effect. At the same time, it can inhibit the effects of PGE1, bradykinin and histamine, increase the pain threshold and produce analgesic effect.

It is a central nervous system stimulant that can excite the cerebral cortex, increase sensitivity to the outside world, and constrict cerebral blood vessels, thereby enhancing the effect of the first two drugs in relieving headaches.

It has a strong histamine H1 receptor blocking effect, can alleviate other respiratory symptoms caused by allergies, and also has a mild inhibitory effect on the central nervous system.

Piperazine has the effect of paralyzing the muscles of roundworms. Its mechanism may be that piperazine exerts an anticholinergic effect at the neuromuscular junction of the worm body, blocks the excitatory effect of acetylcholine on the muscles of roundworms, or changes the permeability of the muscle cell membrane of the worm body to ions, affecting the transmission of nerve impulses; it can also inhibit the production of succinate, reduce the supply of energy, block the neuromuscular junction, and prevent the impulse from being delivered. As a result, the roundworms are detached from the parasitic site and excreted from the body with intestinal peristalsis. Ascaris does not show excitatory effects before paralysis, so it is safer to use this product. This product has no effect on roundworm larvae, and its effect on the muscles of mammals is also very weak. Its mechanism of action on pinworms is not yet clear.

Piperazine has the effect of paralyzing the muscles of roundworms. Its mechanism may be that piperazine exerts an anticholinergic effect at the neuromuscular junction of the worm body, blocks the excitatory effect of acetylcholine on the muscles of roundworms, or changes the permeability of the muscle cell membrane of the worm body to ions, affecting the transmission of nerve impulses; it can also inhibit the production of succinate, reduce the supply of energy, block the neuromuscular junction, and prevent the impulse from being delivered. As a result, the roundworms are detached from the parasitic site and excreted from the body with intestinal peristalsis. Ascaris does not show excitatory effects before paralysis, so it is safer to use this product. This product has no effect on roundworm larvae, and its effect on the muscles of mammals is also very weak. Its mechanism of action on pinworms is not yet clear.