Antiviral drug. It has an inhibitory effect on herpes simplex virus, varicella zoster virus, cytomegalovirus, etc. in vitro. After entering the cells infected by herpes virus, this product competes with deoxynucleoside for viral thymidine kinase or cell kinase, and the drug is phosphorylated into activated acyclovir triphosphate, and then inhibits viral replication in two ways: ① Interfering with viral DNA polymerase and inhibiting viral replication; ② Under the action of DNA polymerase, it binds to the growing DNA chain, causing the extension of the DNA chain to be interrupted. This product has a special affinity for viruses, but has low toxicity to mammalian host cells.

Synthetic nucleoside antiviral drugs, have inhibitory effects on herpes simplex virus type 1 (HSV-1), HSV-2 and varicella-zoster virus (VZV). It is converted into monophosphate by virus-encoded thymidine kinase and further metabolized into triphosphate, which stops the replication of herpes virus DNA. It has the strongest inhibitory effect on HSV-1 virus, followed by HSV-2 and VZV virus.

It has a strong killing effect on the asexual body of malarial parasites and can quickly kill malarial parasites, thereby quickly controlling symptoms.

The antimalarial effect is similar to that of chloroquine. After oral absorption, it is stored in the liver and then slowly released into the blood. It has a long-lasting effect and can be used for the prevention and treatment of malaria symptoms.

It has the effect of inhibiting dihydrofolate reductase. It binds to the dihydrofolate reductase of malarial parasites, interferes with their purine synthesis, and thus inhibits the growth of malarial parasites.

This product is a hypoglycemic drug. It can reduce fasting and postprandial hyperglycemia in patients with type 2 diabetes, and HbAlc can be reduced by 1% to 2%. The mechanism of lowering blood sugar may be: 1. Increasing the sensitivity of peripheral tissues to insulin and increasing insulin-mediated glucose utilization; 2. Increasing the utilization of glucose by non-insulin-dependent tissues, such as the brain, blood cells, renal medulla, intestines, skin, etc.; 3. Inhibiting hepatic gluconeogenesis and reducing hepatic glucose output; 4. Inhibiting intestinal wall cells from taking up glucose; 5. Inhibiting the biosynthesis and storage of cholesterol and reducing blood triglyceride and total cholesterol levels.