It is a natural substance in the body that is essential for mammalian energy metabolism. Its main function is to promote lipid metabolism. In ischemia and hypoxia, it can reduce the accumulation of acyl-CoA, increase free carnitine, maintain the activity of adenine nucleotide translocase, and promote the smooth progress of oxidative phosphorylation. As the main energy source for muscle cells, especially myocardial cells, it can also increase the activity of NADH cell C pigment reductase and cytochrome oxidase, accelerate the production of ATP, and participate in drug detoxification. For various tissue ischemia and hypoxia, it can increase the energy production to improve the energy supply of tissues and organs. At the same time, it has the functions of medium-chain fatty acid oxidation, fatty acid peroxidase oxidation, buffering the ratio of bound coenzyme A and free coenzyme A, generating energy from ketones, pyruvate, amino acids (including branched-chain amino acids), removing the toxicity of excessive coenzyme A, and regulating the ammonia concentration in the blood.

This product belongs to the aminoglycoside antibiotics, and its antibacterial spectrum is similar to that of gentamicin. It has antibacterial effects on a variety of Gram-negative bacteria, and its antibacterial effect on Pseudomonas aeruginosa is 3 to 5 times stronger than that of gentamicin. It is sensitive to Staphylococcus aureus, and resistant to streptococci and anaerobic bacteria, Mycobacterium tuberculosis, Rickettsia, viruses and fungi. The mechanism of action is to bind to the 30S subunit of the bacterial ribosome and inhibit the synthesis of bacterial proteins.

The antiasthmatic effect of this product is similar to that of theophylline, and its cardiac stimulant effect is only 1/20~1/10 of that of aminophylline, and its effect on the heart and nervous system is less. It is especially suitable for asthma patients with tachycardia. This product has a direct relaxant effect on the smooth muscle of the respiratory tract. Its mechanism of action includes inhibiting phosphodiesterase to increase the intracellular cAMP content, releasing endogenous adrenaline and norepinephrine to cause bronchial dilation, acting as a purine receptor blocker to counteract the contractile effect of adenine on the respiratory tract, and enhancing the contractility of the diaphragm to improve respiratory function.

Pharmacological action not specified

It has the effects of ranitidine in inhibiting gastric acid and pepsin secretion, and bismuth citrate in resisting Helicobacter pylori and protecting gastric mucosa.