It has atropine-like parasympathetic inhibitory effect, and can cause mydriasis and accommodation paralysis after drug absorption

It has a sympathetic nerve excitatory effect, and after absorption, it manifests as mydriasis and local vasoconstriction

It works by inhibiting bacterial DNA gyrase and DNA replication. Due to its unique mechanism of action, it has a broad antibacterial spectrum and strong antibacterial activity, and has a strong antibacterial effect on both Gram-negative and Gram-positive bacteria. It is effective against infections caused by Staphylococcus, Streptococcus pyogenes, hemolytic Streptococcus, Enterococcus, Pneumococcus, Escherichia coli, Citrobacter, Klebsiella pneumoniae, Enterobacter, Serratia, Proteus, Pseudomonas aeruginosa, Haemophilus influenzae, Acinetobacter, Campylobacter, and Chlamydia. There is no cross-resistance between this product and other types of antibacterial drugs.

Sodium and chloride are important electrolytes in the body, mainly present in extracellular fluid, and play an important role in maintaining normal blood and extracellular fluid in the human body. For external use, use physiological sodium chloride solution to wash wounds and rinse eyes.

Sodium and chloride are important electrolytes in the body, mainly present in extracellular fluid, and play an important role in maintaining normal blood and extracellular fluid in the human body. For external use, use physiological sodium chloride solution to wash wounds and rinse eyes.

It has a weak local anesthetic effect, can reduce the irritation of irritants, stimulate epithelial proliferation and promote wound healing.

It has astringent and bactericidal effects, and can form a protein film with proteins in inflammatory tissues or secretions, causing capillaries to contract and relieve local congestion.

Inhibits cyclooxygenase activity, thereby blocking the conversion of arachidonic acid to prostaglandins; promotes the combination of arachidonic acid and triglycerides, reduces the concentration of free arachidonic acid in cells, and indirectly inhibits the synthesis of leukotrienes; has a stronger inhibitory effect on prostaglandin synthesis than aspirin and indomethacin; has a strong inhibitory effect on the collapse of the blood-aqueous humor barrier caused by mechanical, chemical, biological and other stimuli; treats postoperative inflammation of cataract surgery, and can reduce the flare and cell count in the anterior chamber; used in patients undergoing radial keratectomy or laser refractive keratectomy, it can relieve postoperative pain and photophobia.

Diclofenac sodium is a non-steroidal anti-inflammatory analgesic derived from phenylacetic acid. Its mechanism of action is to inhibit the activity of cyclooxygenase, thereby blocking the conversion of arachidonic acid to prostaglandins. At the same time, it can also promote the combination of arachidonic acid with triglycerides, reduce the concentration of free arachidonic acid in cells, and indirectly inhibit the synthesis of leukotrienes. Animal experiments have confirmed that prostaglandins are one of the mediators of intraocular inflammation, which can lead to blood-aqueous barrier collapse, vasodilation, increased vascular permeability, leukocyte chemotaxis, non-cholinergic mechanism pupil constriction, etc. Diclofenac sodium is a stronger non-steroidal anti-inflammatory drug. Its inhibitory effect on prostaglandin synthesis is stronger than that of aspirin and indomethacin. Diclofenac sodium eye drops have a strong inhibitory effect on the collapse of the blood-aqueous barrier caused by mechanical, chemical, biological and other stimuli. Clinical studies have shown that 0.1% diclofenac sodium can reduce the flare and cell count in the anterior chamber in the treatment of post-cataract surgery inflammation; it can relieve postoperative pain and photophobia in patients undergoing radial keratectomy or laser refractive keratectomy, and is better than placebo.