It has high antibacterial activity against common aerobic Gram-positive bacteria, such as Staphylococcus aureus (including those resistant to penicillin G), Staphylococcus epidermidis, β-hemolytic Streptococcus, viridans Streptococcus and Streptococcus pneumoniae. It has good antibacterial effect against anaerobic bacteria, including Clostridium diphtheriae, and Clostridium perfringens. It has no activity against Gram-negative bacteria such as Enterococcus, meningococci, Neisseria gonorrhoeae, and Haemophilus influenzae, as well as fungi. There is no cross-resistance between this product and penicillin, chloramphenicol, cephalosporins and tetracyclines, but there is partial cross-resistance with macrolides. It acts on the 50S subunit of the ribosome of sensitive bacteria, preventing the extension of the peptide chain, thereby inhibiting the protein synthesis of bacterial cells. It is generally an antibacterial agent, but at high concentrations, it also has a bactericidal effect on certain bacteria.

Its effect is similar to or slightly weaker than that of atropine. It has obvious peripheral anticholinergic effect, can relax smooth muscles caused by spasm caused by acetylcholine, relieve spasm of blood vessels (especially microvessels), and improve microcirculation.

It is an acidic mucopolysaccharide extracted and prepared from animal tissues. It carries a large amount of negative charge in the molecule and plays an important role in maintaining the relative stability of the cell environment and normal function.

Competitive antagonist of the agonist effect of acetylcholine or cholinergic receptor agonists on M cholinergic receptors. It has a high selectivity for M receptors, and large or toxic doses can also block the N1 receptors of the ganglion. Eye tissue: Blocks the M cholinergic receptors, thereby relaxing the pupillary sphincter and ciliary muscle, resulting in pupil dilation.

Competitive antagonist of the agonist effect of acetylcholine or cholinergic receptor agonists on M cholinergic receptors. It has a high selectivity for M receptors, and large or toxic doses can also block the N1 receptors of the ganglion. Eye tissue: Blocks the M cholinergic receptors, thereby relaxing the pupillary sphincter and ciliary muscle, resulting in pupil dilation.

Competitive antagonist of the agonist effect of acetylcholine or cholinergic receptor agonists on M cholinergic receptors. It has a high selectivity for M receptors, and large or toxic doses can also block the N1 receptors of the ganglion. Eye tissue: Blocks the M cholinergic receptors, thereby relaxing the pupillary sphincter and ciliary muscle, resulting in pupil dilation.

Competitive antagonist of the agonist effect of acetylcholine or cholinergic receptor agonists on M cholinergic receptors. It has a high selectivity for M receptors, and large or toxic doses can also block the N1 receptors of the ganglion. Eye tissue: Blocks the M cholinergic receptors, thereby relaxing the pupillary sphincter and ciliary muscle, resulting in pupil dilation.

Competitive antagonist of the agonist effect of acetylcholine or cholinergic receptor agonists on M cholinergic receptors. It has a high selectivity for M receptors, and large or toxic doses can also block the N1 receptors of the ganglion. Eye tissue: Blocks the M cholinergic receptors, thereby relaxing the pupillary sphincter and ciliary muscle, resulting in pupil dilation.

Inhibits cyclooxygenase activity, thereby blocking the conversion of arachidonic acid to prostaglandins, while promoting the combination of arachidonic acid and triglycerides, reducing the concentration of free arachidonic acid in cells, and indirectly inhibiting the synthesis of leukotrienes. Its inhibitory effect on prostaglandin synthesis is stronger than that of aspirin and indomethacin, and it has a strong inhibitory effect on the collapse of the blood-aqueous humor barrier caused by mechanical, chemical, biological and other stimuli, and can reduce the flare and cell count in the anterior chamber, and relieve postoperative pain and photophobia.

Antiviral drug. It has the effect of inhibiting the growth of many viruses such as respiratory syncytial virus, influenza virus, hepatitis A virus, adenovirus, etc. in vitro, and its mechanism is not fully understood. This product does not change the adsorption, invasion and uncoating of viruses, nor does it induce the production of interferon. After entering the virus-infected cells, the drug is rapidly phosphorylated, and its product acts as a competitive inhibitor of viral synthase, inhibiting inosine monophosphate dehydrogenase, influenza virus RNA polymerase and mRNA guanosine transferase, thereby causing a decrease in intracellular guanosine triphosphate, impairing viral RNA and protein synthesis, and inhibiting viral replication and transmission. It may also have an immune effect and neutralizing antibody effect on respiratory syncytial virus.