It is a sulfonamide diuretic that works by inhibiting the reabsorption of water and electrolytes in the dilution segment of the distal renal tubular cortex. Possible mechanisms of antihypertensive effects include regulating calcium influx into vascular smooth muscle cells; stimulating the synthesis of prostaglandins PGE2 and prostaglandins PGI2; reducing vascular hypersensitivity to vasopressors, thereby inhibiting vasoconstriction. This product has little or no effect on cardiac output, heart rate and rhythm when lowering blood pressure. Long-term use of this product rarely affects glomerular filtration rate or renal blood flow. This drug does not affect the metabolism of blood lipids and carbohydrates.

It is a sulfonamide diuretic that works by inhibiting the reabsorption of water and electrolytes in the dilution segment of the distal renal tubular cortex. Possible mechanisms of antihypertensive effects include regulating calcium influx into vascular smooth muscle cells; stimulating the synthesis of prostaglandins PGE2 and prostaglandins PGI2; reducing vascular hypersensitivity to vasopressors, thereby inhibiting vasoconstriction. This product has little or no effect on cardiac output, heart rate and rhythm when lowering blood pressure. Long-term use of this product rarely affects glomerular filtration rate or renal blood flow. This drug does not affect the metabolism of blood lipids and carbohydrates.

It is a β2 receptor agonist, has a significant dilating effect on the bronchi, has a weaker antiasthmatic effect, but significantly reduces cardiac toxicity. It is well absorbed orally, and its effective concentration in the blood can be maintained for 6 hours.

It has a strong effect of dissolving mucus and phlegm, which can break down the polysaccharide cellulose in phlegm and thin the phlegm. It inhibits the synthesis of glycoproteins by goblet cells and mucous glands, reduces the sialic acid in phlegm, reduces the viscosity of phlegm, and makes it easier to cough up.

It has antihistamine effect, and has antiasthmatic and sedative effects.

It has a direct relaxing effect on the smooth muscles of the respiratory tract. Its bronchodilator effect is partly due to the promotion of the release of endogenous adrenaline and norepinephrine. It can counteract the contractile effect of adenine on the respiratory tract, enhance the contractility of the diaphragm, and improve respiratory function.

It has the effect of dissolving mucus and phlegm, acting on the mucous gland cells of the trachea and bronchus to secrete low-viscosity small-molecule mucins, which reduces and dilutes the mucus and makes it easier to cough up. It also promotes the movement of respiratory mucosal cilia and has a nauseating expectorant effect.

It is a histamine H1 receptor blocker that can counteract allergic symptoms such as microvascular dilation and increased capillary permeability caused by histamine, and has a mild sedative and M cholinergic receptor blocking effect.

It can increase the activity of microsomal enzymes in liver cells and accelerate the liver's detoxification ability; it can reduce the increase in serum glutamic-alanine aminotransferase in rats caused by carbon tetrachloride; it can stabilize the cell membranes of various types of liver damage caused by liver poisons such as carbon tetrachloride, phalloidin, thioacetamide, and crotalin, achieving a significant liver protection effect.