After entering the body, this product can combine with poisons containing hydroxyl or carboxyl groups to form low-toxic or non-toxic combinations that are excreted in the urine, protecting the liver and detoxifying. In addition, glucuronic acid can increase the content of liver glycogen and reduce fat reserves.

Like vitamin B1, active thiamine combines with pyrophosphate in the body to form a cocarboxylase, which participates in the oxidative decarboxylation reaction of pyruvate and alpha-ketoglutaric acid in sugar metabolism and is necessary for sugar metabolism. When it is deficient, oxidation is blocked to form pyruvate, lactic acid accumulates, and affects the body's energy supply. Its symptoms are mainly manifested in the nervous and cardiovascular systems, with multiple peripheral neuritis affecting both sensory nerves and motor nerves, manifested as paresthesia, neuralgia, limb weakness, and muscle soreness and atrophy. In terms of cardiovascular system, due to the increase of acetone and lactic acid in the blood, the arterioles are dilated, the diastolic pressure is reduced, and the myocardial metabolism is disturbed, so it is easy to have symptoms of heart failure such as palpitations, shortness of breath, chest tightness, cardiac hypertrophy, liver and lung congestion and peripheral edema. In terms of the digestive tract, it is manifested as decreased appetite.

As a histamine H1 receptor antagonist, it can counteract the capillary dilation caused by allergic reactions, reduce the permeability of capillaries, and relieve wheezing caused by bronchial smooth muscle contraction. It has a long-lasting antihistamine effect and also has a significant central inhibitory effect, which can increase the effects of anesthetics, analgesics, hypnotics and local anesthetics. This product is mainly metabolized in the liver.

Like vitamin B1, active thiamine combines with pyrophosphate in the body to form a cocarboxylase, which participates in the oxidative decarboxylation reaction of pyruvate and alpha-ketoglutaric acid in sugar metabolism and is necessary for sugar metabolism. When it is deficient, oxidation is blocked to form pyruvate, lactic acid accumulates, and affects the body's energy supply. Its symptoms are mainly manifested in the nervous and cardiovascular systems, with multiple peripheral neuritis affecting both sensory nerves and motor nerves, manifested as paresthesia, neuralgia, limb weakness, and muscle soreness and atrophy. In terms of cardiovascular system, due to the increase of acetone and lactic acid in the blood, the arterioles are dilated, the diastolic pressure is reduced, and the myocardial metabolism is disturbed, so it is easy to have symptoms of heart failure such as palpitations, shortness of breath, chest tightness, cardiac hypertrophy, liver and lung congestion and peripheral edema. In terms of the digestive tract, it is manifested as decreased appetite.

Rich in B vitamins, it has an auxiliary therapeutic effect on indigestion