Valsartan is an orally effective specific angiotensin (AT) II receptor antagonist that selectively acts on the AT1 receptor subtype and has an affinity for the AT1 receptor that is 20,000 times stronger than that for the AT2 receptor. Valsartan has no inhibitory effect on ACE and does not cause retention of bradykinin or substance P, so it does not cause coughing. Valsartan lowers elevated blood pressure without affecting heart rate.

Nonsteroidal anti-inflammatory drugs that inhibit the biosynthesis of prostaglandins, and their biological activity is related to their S-form. Animal studies have shown analgesic effects, but no sedative or antianxiety effects.

Analgesic effect: It may also have a central analgesic effect by inhibiting the synthesis of prostaglandins and other sensitive substances; Anti-inflammatory effect: It may inhibit the synthesis of prostaglandins or other inflammatory substances, and may also be related to the inhibition of lysosomal enzyme release and leukocyte chemotaxis; Antipyretic effect: It may act on the hypothalamic temperature regulation center to cause increased heat dissipation, and this effect may be related to the inhibition of prostaglandin synthesis in the hypothalamus; Anti-rheumatic effect: In addition to antipyretic and analgesic effects, it mainly has anti-inflammatory effects; Inhibition of platelet aggregation: It reduces prostaglandin production by inhibiting platelet cyclooxygenase.

This product is an azole antifungal drug, which has antibacterial effects on Candida, Chromatiaceae, Coccidioides, Histoplasma, Sporothrix, etc., and also has antibacterial activity against Trichophyton, etc. It interferes with the activity of cytochrome P-450, inhibits the biosynthesis of ergosterol, the main steroid of fungal cell membrane, damages the fungal cell membrane and changes its permeability, leading to leakage of important intracellular substances. It can inhibit the biosynthesis of triglycerides and phospholipids of fungi, inhibit the activity of oxidase and peroxidase, cause the accumulation of hydrogen peroxide in cells, and lead to cell submicrostructure degeneration and cell necrosis. For Candida albicans, it can inhibit the process of transformation from spores to invasive hyphae.

This product is an azole antifungal drug and a dechlorinated derivative of miconazole. This product has antibacterial effects on Candida, Chromatiaceae, Coccidioides, Histoplasma, Sporothrix, etc., and also has antibacterial activity against Trichophyton, etc. This product has poor effects on Aspergillus, Sporothrix Schenckii, some dark-colored spores, Mucor, etc. This product inhibits the biosynthesis of ergosterol, the main steroid of fungal cell membrane, by interfering with the activity of cytochrome P-450, damaging the fungal cell membrane and changing its permeability, resulting in leakage of important intracellular substances. This product can inhibit the biosynthesis of triglycerides and phospholipids of fungi, inhibit the activity of oxidases and peroxidases, cause the accumulation of hydrogen peroxide in cells, and lead to cell submicrostructure degeneration and cell necrosis. For Candida albicans, it can inhibit the process of transformation from spores to invasive hyphae.