It binds to the 50S subunit of bacterial ribosomes, preventing the elongation of peptide chains, thereby inhibiting protein synthesis in bacterial cells. It has high antibacterial activity against Gram-positive bacteria such as Staphylococcus (including penicillin-resistant strains), Streptococcus, Corynebacterium diphtheriae, Bacillus anthracis, etc. It also has good antibacterial activity against Gram-negative anaerobic bacteria, and most of the Bacteroides, including Bacteroides fragilis, Fusobacterium, Peptococcus, Peptostreptococcus, and Clostridium perfringens, are highly sensitive. It is resistant to Gram-negative aerobic bacteria including Haemophilus influenzae, Neisseria, and Mycoplasma. It has a bactericidal effect on certain bacteria at high concentrations.

It can reduce the level of DHBV-DNA in the serum of ducks infected with hepatitis B virus (DHBV) and has a protective effect on toxic liver damage in mice caused by CCL4 and galactosamine

It can reduce the level of DHBV-DNA in the serum of ducks infected with hepatitis B virus (DHBV) and has a protective effect on toxic liver damage in mice caused by CCL4 and galactosamine

It can reduce the level of DHBV-DNA in the serum of ducks infected with hepatitis B virus (DHBV) and has a protective effect on toxic liver damage in mice caused by CCL4 and galactosamine

It forms thiamine pyrophosphate in the body and is a necessary coenzyme for carbohydrate metabolism, which can maintain the normal functions of the heart, nerves and digestive system. It can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, leading to nerve impulse conduction disorders and affecting gastrointestinal and myocardial functions.

It is converted into pyridoxal phosphate in red blood cells and acts as a coenzyme in various metabolic functions of proteins, carbohydrates, and lipids. It is also involved in the conversion of tryptophan into niacin or 5-hydroxytryptamine.

It participates in the methyl conversion and folic acid metabolism in the body, promotes the conversion of 5-methyltetrahydrofolate into tetrahydrofolate, promotes the conversion of methylmalonic acid into succinic acid, and participates in the tricarboxylic acid cycle. This effect is related to the synthesis of nerve myelin sphingolipids and the maintenance of the integrity of spinal nerve fiber function. The nerve damage caused by deficiency may be related to this.