Sichuan Guorui Pharmaceutical Co., Ltd.

It is a similar substance of aminobutyric acid, which can activate, protect and repair brain cells, improve brain hypoxia, activate brain cells, increase the ATP/ADP ratio in the brain, promote the absorption of amino acids and phospholipids, protein synthesis, glucose utilization and energy storage, promote brain metabolism, increase cerebral blood flow. It can accelerate the speed of information transmission between the cerebral hemispheres through the corpus callosum, and improve the ability of learning, memory and thinking activities.

This product cannot be taken at the same time as enteric-coated tablets. If used at the same time with other drugs, drug interactions may occur. Please consult your doctor or pharmacist for details.

This product cannot be taken at the same time as enteric-coated tablets. If used at the same time with other drugs, drug interactions may occur. Please consult your doctor or pharmacist for details.

It inhibits the synthesis and release of prostaglandins in the hypothalamus, restores the normal responsiveness of the sensory neurons in the temperature regulation center, and has an antipyretic effect; it has an analgesic effect by inhibiting the synthesis of prostaglandins, etc.; it inhibits the synthesis and release of prostaglandins in the local tissue of inflammation, stabilizes lysosomal enzymes, and affects the phagocytic function of phagocytes, thus playing an anti-inflammatory role

It is a peripheral analgesic. It inhibits the synthesis and release of prostaglandins (PGE1) in the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating to achieve an antipyretic effect; it can also inhibit the effects of PGE1, bradykinin and histamine, increase the pain threshold and produce analgesic effects.

It is a central nervous system stimulant that can excite the cerebral cortex, increase sensitivity to the outside world, and constrict cerebral blood vessels, thus enhancing the headache relief effects of the first two drugs.

It has a strong histamine H1 receptor blocking effect, can alleviate other respiratory symptoms caused by allergies, and also has a mild inhibitory effect on the central nervous system

It exerts a sedative and hypnotic effect by activating the GABA receptor-chloride ion complex; increases chloride ion conduction, inhibits the central nervous system, and produces sedative and hypnotic effects; the anesthetic potency is 1.8 times that of thiopental sodium; it takes effect quickly, has a short duration of action, and awakens quickly; it can inhibit the throat reflex, reduce intracranial pressure and intraocular pressure, and reduce brain oxygen consumption and cerebral blood flow; postoperative nausea and vomiting are rare, and the analgesic effect is very weak; it has a synergistic effect when used with other central nervous system depressants; it can cause a drop in blood pressure and respiratory depression; involuntary muscle movements and convulsions occur during anesthesia induction.

It exerts a sedative and hypnotic effect by activating the GABA receptor-chloride ion complex. It increases chloride ion conduction, desensitizes GABA receptors at high doses, inhibits the central nervous system, produces sedative and hypnotic effects, and its anesthetic potency is 1.8 times that of sodium thiopental. It has a fast onset and a short duration of action. When injected intravenously at 2.5 mg/kg, the onset time is 30 to 60 seconds, and the duration is about 10 minutes. It wakes up quickly and there is no hangover feeling after waking up. It can inhibit the throat reflex, which is conducive to intubation, and laryngospasm rarely occurs. It reduces intracranial pressure and intraocular pressure, reduces brain oxygen consumption and cerebral blood flow, and postoperative nausea and vomiting are rare, and the analgesic effect is very weak. It has a synergistic effect when used in combination with other central nervous system depressants. The use of propofol can reduce the plasma corticosteroid concentration, but the adrenal cortex responds normally to exogenous corticosteroids. Involuntary muscle movements and convulsions occur during anesthesia induction, which are more obvious during shallow anesthesia.

1. This product exerts a sedative and hypnotic effect by activating the GABA receptor-chloride ion complex. At clinical doses, propofol increases chloride ion conduction, and at high doses, it desensitizes GABA receptors, thereby inhibiting the central nervous system and producing sedative and hypnotic effects. Its anesthetic potency is 1.8 times that of sodium thiopental. It has a fast onset and a short duration of action. When injected intravenously at 2.5 mg/kg, the onset time is 30 to 60 seconds, and the duration is about 10 minutes. It wakes up quickly and there is no hangover feeling after waking up. 2. It can inhibit the throat reflex, facilitate intubation, and rarely cause laryngeal spasm. It has an inhibitory effect on the circulatory system. When this product is used for general anesthesia induction, it can cause a drop in blood pressure, a decrease in myocardial blood perfusion and oxygen consumption, a decrease in peripheral vascular resistance, and no significant change in heart rate. Propofol can reduce blood pressure, and the degree of reduction exceeds 40% in some patients. It is particularly obvious for the elderly and weak, and patients with heart failure to reduce blood pressure. The dose should be reduced as appropriate, and the intravenous injection speed should be slowed down. Propofol also has a significant inhibitory effect on respiration and can inhibit the ventilatory response to carbon dioxide, which is manifested as a decrease in tidal volume. It can increase the respiratory rate when awake, and apnea often occurs after intravenous injection. It has no significant effect on bronchial smooth muscle. 3. Propofol can reduce intracranial pressure and intraocular pressure, reduce cerebral oxygen consumption and cerebral blood flow, and postoperative nausea and vomiting are rare, and the analgesic effect is very weak. It has a synergistic effect when used in combination with other central nervous system depressants. The use of propofol can reduce plasma corticosteroid concentrations, but the adrenal cortex responds normally to exogenous corticosteroids. Propofol anesthesia induction produces involuntary muscle movements and convulsions, which are more obvious during light anesthesia.

1. This product exerts a sedative and hypnotic effect by activating the GABA receptor-chloride ion complex. At clinical doses, propofol increases chloride ion conduction, and at high doses, it desensitizes GABA receptors, thereby inhibiting the central nervous system and producing sedative and hypnotic effects. Its anesthetic potency is 1.8 times that of sodium thiopental. It has a fast onset and a short duration of action. When injected intravenously at 2.5 mg/kg, the onset time is 30 to 60 seconds, and the duration is about 10 minutes. It wakes up quickly and there is no hangover feeling after waking up. 2. It can inhibit the throat reflex, facilitate intubation, and rarely cause laryngeal spasm. It has an inhibitory effect on the circulatory system. When this product is used for general anesthesia induction, it can cause a drop in blood pressure, a decrease in myocardial blood perfusion and oxygen consumption, a decrease in peripheral vascular resistance, and no significant change in heart rate. Propofol can reduce blood pressure, and the degree of reduction exceeds 40% in some patients. It is particularly obvious for the elderly and weak, and patients with heart failure to reduce blood pressure. The dose should be reduced as appropriate, and the intravenous injection speed should be slowed down. Propofol also has a significant inhibitory effect on respiration and can inhibit the ventilatory response to carbon dioxide, which is manifested as a decrease in tidal volume. It can increase the respiratory rate when awake, and apnea often occurs after intravenous injection. It has no significant effect on bronchial smooth muscle. 3. Propofol can reduce intracranial pressure and intraocular pressure, reduce cerebral oxygen consumption and cerebral blood flow, and postoperative nausea and vomiting are rare, and the analgesic effect is very weak. It has a synergistic effect when used in combination with other central nervous system depressants. The use of propofol can reduce plasma corticosteroid concentrations, but the adrenal cortex responds normally to exogenous corticosteroids. Propofol anesthesia induction produces involuntary muscle movements and convulsions, which are more obvious during light anesthesia.

1. This product exerts a sedative and hypnotic effect by activating the GABA receptor-chloride ion complex. At clinical doses, propofol increases chloride ion conduction, and at high doses, it desensitizes GABA receptors, thereby inhibiting the central nervous system and producing sedative and hypnotic effects. Its anesthetic potency is 1.8 times that of sodium thiopental. It has a fast onset and a short duration of action. When injected intravenously at 2.5 mg/kg, the onset time is 30 to 60 seconds, and the duration is about 10 minutes. It wakes up quickly and there is no hangover feeling after waking up. 2. It can inhibit the throat reflex, facilitate intubation, and rarely cause laryngeal spasm. It has an inhibitory effect on the circulatory system. When this product is used for general anesthesia induction, it can cause a drop in blood pressure, a decrease in myocardial blood perfusion and oxygen consumption, a decrease in peripheral vascular resistance, and no significant change in heart rate. Propofol can reduce blood pressure, and the degree of reduction exceeds 40% in some patients. It is particularly obvious for the elderly and weak, and patients with heart failure to reduce blood pressure. The dose should be reduced as appropriate, and the intravenous injection speed should be slowed down. Propofol also has a significant inhibitory effect on respiration and can inhibit the ventilatory response to carbon dioxide, which is manifested as a decrease in tidal volume. It can increase the respiratory rate when awake, and apnea often occurs after intravenous injection. It has no significant effect on bronchial smooth muscle. 3. Propofol can reduce intracranial pressure and intraocular pressure, reduce cerebral oxygen consumption and cerebral blood flow, and postoperative nausea and vomiting are rare, and the analgesic effect is very weak. It has a synergistic effect when used in combination with other central nervous system depressants. The use of propofol can reduce plasma corticosteroid concentrations, but the adrenal cortex responds normally to exogenous corticosteroids. Propofol anesthesia induction produces involuntary muscle movements and convulsions, which are more obvious during light anesthesia.

1. This product exerts a sedative and hypnotic effect by activating the GABA receptor-chloride ion complex. At clinical doses, propofol increases chloride ion conduction, and at high doses, it desensitizes GABA receptors, thereby inhibiting the central nervous system and producing sedative and hypnotic effects. Its anesthetic potency is 1.8 times that of sodium thiopental. It has a fast onset and a short duration of action. When injected intravenously at 2.5 mg/kg, the onset time is 30 to 60 seconds, and the duration is about 10 minutes. It wakes up quickly and there is no hangover feeling after waking up. 2. It can inhibit the throat reflex, facilitate intubation, and rarely cause laryngeal spasm. It has an inhibitory effect on the circulatory system. When this product is used for general anesthesia induction, it can cause a drop in blood pressure, a decrease in myocardial blood perfusion and oxygen consumption, a decrease in peripheral vascular resistance, and no significant change in heart rate. Propofol can reduce blood pressure, and the degree of reduction exceeds 40% in some patients. It is particularly obvious for the elderly and weak, and patients with heart failure to reduce blood pressure. The dose should be reduced as appropriate, and the intravenous injection speed should be slowed down. Propofol also has a significant inhibitory effect on respiration and can inhibit the ventilatory response to carbon dioxide, which is manifested as a decrease in tidal volume. It can increase the respiratory rate when awake, and apnea often occurs after intravenous injection. It has no significant effect on bronchial smooth muscle. 3. Propofol can reduce intracranial pressure and intraocular pressure, reduce cerebral oxygen consumption and cerebral blood flow, and postoperative nausea and vomiting are rare, and the analgesic effect is very weak. It has a synergistic effect when used in combination with other central nervous system depressants. The use of propofol can reduce plasma corticosteroid concentrations, but the adrenal cortex responds normally to exogenous corticosteroids. Propofol anesthesia induction produces involuntary muscle movements and convulsions, which are more obvious during light anesthesia.