1. Treat metabolic acidosis. This product increases the concentration of carbonate in plasma and neutralizes hydrogen ions, thereby correcting acidosis. 2. Alkalinize urine. As the pH value increases after the concentration of bicarbonate in urine increases, uric acid, sulfonamide drugs and hemoglobin are not easy to form crystals or aggregate in urine. 3. Antacid. Oral administration can quickly neutralize or buffer gastric acid without directly affecting gastric acid secretion. Therefore, the pH in the stomach increases rapidly to relieve the symptoms caused by high gastric acid.

It has a direct relaxant effect on the smooth muscles of the respiratory tract, inhibits phosphodiesterase to increase the intracellular cAMP content, releases endogenous adrenaline and norepinephrine to cause bronchial dilation, acts as a purine receptor blocker to counteract the contractile effect of adenine on the respiratory tract, enhances the contractility of the diaphragm and improves respiratory function, slightly relaxes the peripheral blood vessels of the coronary arteries and bile duct smooth muscles, slightly increases contractility and has a slight diuretic effect.

It has a strong affinity for steroid metabolic enzymes in the liver, thereby hindering the inactivation of cortisol and aldosterone, showing obvious corticosteroid-like effects, such as anti-inflammatory, anti-allergic and protective membrane structures; promoting monochromatic metabolism, reducing the release of ALT and AST; regulating calcium ion channels, protecting lysosomal membranes and mitochondria

Induce γ-IFN and interleukin Ⅱ, increase NK cell activity and OKT4/OKT8 ratio and activate the reticuloendothelial system; inhibit mast cell release of histamine; inhibit the production and formation of free radicals and lipid peroxides, and reduce the activity of proline hydroxylase

Inhibits the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reaction

It has a strong affinity for steroid metabolic enzymes in the liver, thereby hindering the inactivation of cortisol and aldosterone, showing obvious corticosteroid-like effects, such as anti-inflammatory, anti-allergic and protective membrane structures; promoting monochromatic metabolism, reducing the release of ALT and AST; inducing γ-IFN and interleukin II, increasing NK cell activity and OKT4/OKT8 ratio and activating the reticuloendothelial system; regulating calcium ion channels, protecting lysosomal membranes and mitochondria

Inhibit the release of histamine from mast cells; inhibit the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reaction; inhibit the production and formation of free radicals and lipid peroxides, and reduce the activity of proline hydroxylase

No special mention of pharmacological effects

This product is a coenzyme that improves the body's metabolism and participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotides in the body. It is also the main source of energy in the body. When energy is needed for absorption, secretion, muscle contraction and biochemical synthesis reactions in the body, adenosine triphosphate is decomposed into adenosine diphosphate and phosphate groups, and energy is released at the same time. Animal experiments have shown that this product can inhibit the slow calcium ion influx of slow-reacting fibers, block or delay the forward conduction in the atrioventricular node reentry pathway, and a large dose may also block or delay the forward and reverse conduction of the bypass; in addition, it also has a short-term and strong effect of enhancing the vagus nerve, thereby terminating arrhythmias caused by atrioventricular node reentry and bypass reentry mechanisms.