Jilin Xianfeng Technical Pharmaceutical Co., Ltd.

This product is a mild peripheral vasodilator, which is gradually hydrolyzed into niacin and inositol in the body, so it has the pharmacological effects of both niacin and inositol, and has lipid-lowering effects. Its vasodilating effect is milder and more lasting than that of niacin, and it does not have side effects such as flushing and stomach discomfort after taking niacin. This product can selectively dilate blood vessels in lesions and sensitive areas stimulated by cold, while its dilating effect on normal blood vessels is weaker. In addition, it has the effects of dissolving blood clots, anticoagulation, anti-fatty liver, and reducing capillary fragility.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

It has a significant antihypertensive effect on renal hypertension; it can counteract arrhythmias caused by aconitine, calcium chloride and coronary artery ligation. Its antiarrhythmic mechanism may be related to affecting calcium ion influx.

1. Effects on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc.; by inhibiting the active reabsorption of NaCl by the thick wall segment of the renal tubular loop, it reduces the osmotic pressure gradient difference of the medullary interstitial fluid, resulting in increased excretion of water, Na, and Cl-; it promotes the secretion of K by the distal tubules; it inhibits the reabsorption of Ca2 and Mg2 by the loop of Henle and increases their excretion; short-term medication can increase uric acid excretion, while long-term medication can cause hyperuricemia. 2. Effects on hemodynamics: It inhibits the activity of prostaglandin degrading enzymes, increases the content of prostaglandin E2, and dilates blood vessels; it reduces renal vascular resistance and increases renal blood flow; it does not reduce the glomerular filtration rate while diuresis; it dilates the pulmonary capacitance veins, reduces the permeability of pulmonary capillaries, reduces the amount of blood returning to the heart, and reduces the left ventricular end-diastolic pressure, which is helpful for the treatment of acute left heart failure.

It can inhibit the synthesis and release of prostaglandin E1 in the neurons of the anterior hypothalamus, restore the normal responsiveness of the sensory neurons in the temperature regulation center, and play an antipyretic role; it can also inhibit the synthesis and release of prostaglandins in the local tissue of inflammation, stabilize the lysosomal membrane, and affect the phagocytic function of phagocytes, thereby playing an anti-inflammatory role.

As a central nervous system stimulant, it can excite the cerebral cortex, increase sensitivity to the outside world, and constrict cerebral blood vessels to enhance the effect of aminopyrine in relieving headaches, and synergistically relieve fever and analgesia.

It has pharmacological effects such as sedation, anticonvulsant, enhancing hypoxia tolerance, and enhancing the body's immune function.

Honey fungus has similar pharmacological effects to Gastrodia elata. Honey fungus fruiting bodies contain mannitol, lecithin, ergosterol, vitamin A (high content), vitamin B, C and various amino acids. It has pharmacological effects such as sedation, anticonvulsant, enhancing hypoxia tolerance, and enhancing the body's immune function.