A mild peripheral vasodilator with the pharmacological effects of niacin and inositol, it can lower blood lipids; the vasodilator effect is mild and long-lasting, without the side effects of niacin such as flushing and stomach discomfort; it selectively dilates blood vessels in diseased areas and areas sensitive to cold stimulation; it has a weaker dilation effect on normal blood vessels; it has the effects of dissolving blood clots, anti-coagulation, anti-fatty liver, and reducing capillary fragility.

This product has a broad-spectrum antimicrobial effect in vitro, including aerobic Gram-negative and Gram-positive bacteria, anaerobes, Rickettsia, spirochetes and Chlamydia. It has bactericidal effects on the following bacteria: Haemophilus influenzae, Streptococcus pneumoniae and Neisseria meningitidis. It only has antibacterial effects on the following bacteria: Staphylococcus aureus, Streptococcus pyogenes, Streptococcus viridans, Group B hemolytic Streptococcus, Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Salmonella typhi, Salmonella paratyphi, Shigella, Bacteroides fragilis and other anaerobic bacteria. The following bacteria are usually resistant to chloramphenicol: Pseudomonas aeruginosa, Acinetobacter, Enterobacter, Serratia marcescens, indole-positive Proteus, methicillin-resistant Staphylococcus and Enterococcus. This product is an antibacterial agent. Chloramphenicol is fat-soluble and enters bacterial cells by diffusion, and reversibly binds to the 50S subunit of the bacterial ribosome, which blocks the growth of the peptide chain (possibly due to the inhibition of the action of transpeptidase), thereby inhibiting the formation of the peptide chain and preventing protein synthesis.

This product is a gastrin receptor antagonist, which can significantly inhibit the secretion of gastric acid and pepsin caused by gastrin, increase the content of hexosamine in gastric mucosa, promote glycoprotein synthesis, and protect and promote the healing of gastric mucosa. In addition, this product has a choleretic effect and can prevent the formation of gallstones by stimulating bile acid-independent bile secretion, changing the stone-forming factors in bile, and antagonizing CCK. The use of this product to treat peptic ulcers and gastritis does not cause rebound gastric acid secretion, and gastric acid secretion can remain at a normal level for up to half a year after the treatment is terminated.

This product is a broad-spectrum antibacterial agent, with bactericidal effect at high concentration. It has antibacterial effect on Streptococcus pneumoniae, hemolytic Streptococcus, viridans Streptococcus and some Staphylococci, Clostridium perfringens, Bacillus anthracis, Yersinia pestis, diphtheria, tetanus, Brucella, Haemophilus influenzae, Campylobacter, Vibrio cholerae, etc. It also has inhibitory effect on Rickettsia, Mycoplasma, Chlamydia, spirochetes, and some protozoa. It binds to the A position of the 30S subunit of the bacterial ribosome, preventing the connection of aminoacyl-tRNA at this position, thereby inhibiting the growth of peptide chains and affecting the synthesis of bacterial proteins.