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Aminoglycoside antibiotics have antibacterial effects on most Enterobacteriaceae, Pseudomonas aeruginosa, some other Pseudomonas, Acinetobacter, Alcaligenes, meningococci, gonococci, influenza bacilli, Yersinia, Campylobacter fetus, Mycobacterium tuberculosis and some Mycobacterium. The mechanism of action is to act on the 30S subunit of bacterial ribosomes and inhibit bacterial protein synthesis. Combination with semi-synthetic penicillins or cephalosporins often produces synergistic antibacterial effects.

It is a nucleoside diphosphate reductase inhibitor that can prevent nucleotides from being reduced to deoxynucleotides, interfere with the biosynthesis of purine and pyrimidine bases, selectively inhibit DNA synthesis, and have no blocking effect on RNA and protein synthesis. It is a cycle-specific drug, and is sensitive to S-phase cells.

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Granulocyte stimulating factor binds to the surface receptors of hematopoietic cells and acts on hematopoietic cells, thereby stimulating proliferation, differentiation, typing and activation of mature cell functions. Compared with rhG-CSF, PEG-rhG-CSF can reduce plasma clearance rate and prolong half-life.