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This product is a lipid-regulating drug of the clofibric acid derivative class. It achieves the purpose of lowering blood lipids by reducing the amount of very low-density lipoprotein, which may involve inhibiting the release of liver lipoprotein (especially very low-density lipoprotein) and cholesterol synthesis, changing the synthesis of triglycerides in the liver, enhancing the action of lipoprotein esterase, increasing the secretion of steroids and their excretion from the feces, and increasing the clearance of triglycerides (very low-density lipoprotein) in the circulation; the anti-diabetes insipidus effect of this product comes from the fact that this product stimulates the posterior pituitary to release antidiuretic hormone.

This product belongs to the butyrophenone antipsychotic drug. Its antipsychotic effect is related to its ability to block dopamine receptors in the brain and promote the conversion of dopamine in the brain. It has a good effect on resisting hallucinations and delusions and excitement and agitation, has a strong effect in blocking extrapyramidal dopamine, and also has a strong antiemetic effect, but its sedative and α-adrenaline receptor and cholinergic receptor blocking effects are weak.

1. Nifedipine is a dihydropyridine calcium antagonist that can selectively inhibit the transmembrane transport of calcium ions into myocardial cells and smooth muscle cells, and inhibit the release of calcium ions from intracellular reservoirs without changing the plasma calcium ion concentration. 2. It can simultaneously relax the coronary arteries in the normal blood supply area and the ischemic area, antagonize spontaneous or ergonovine-induced coronary artery spasm, increase the delivery of myocardial oxygen to patients with coronary artery spasm, and relieve and prevent coronary artery spasm. It can also inhibit myocardial contraction, reduce myocardial metabolism, and reduce myocardial oxygen consumption. On the other hand, it can relax peripheral resistance vessels, reduce peripheral resistance, reduce systolic and diastolic blood pressure, and reduce cardiac afterload. 3. It can delay the sinoatrial node function and atrioventricular conduction of isolated hearts. Electrophysiological studies of whole animals and humans have not found that this product has the effect of delaying atrioventricular conduction, prolonging the recovery time of the sinoatrial node, and slowing down the sinoatrial node rate.

Participates in the formation of bones and the reconstruction of bone tissue after fracture, muscle contraction, nerve transmission, coagulation process and reduces the permeability of capillaries, etc.