Shandong Shenglu Pharmaceutical Co., Ltd.
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Founded in:
2001-12-27 -
Country:
China -
Address:
North end of Sihe Road, Sihe Office, Sishui County -
Tax NO.:
91370831678104460B -
Registered Funds:
1.2 million yuan -
Website:
-
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| DL-Methionine |
Methionine is one of the eight essential amino acids for the human body. It cannot be synthesized in the human body and must be supplemented from outside. Methionine combines with ATP in the human body to generate S-adenosine. It can promote the methylation of liver cell membrane phospholipids, reduce intrahepatic bile stasis, and enhance the transsulfurization effect. It is beneficial for liver cells to restore normal physiological functions, promote the disappearance of jaundice and the recovery of liver function. By supplying methyl groups, it promotes the synthesis of choline, promotes liver fat metabolism, protects the liver and detoxifies.
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Methionine is one of the eight essential amino acids for the human body. It cannot be synthesized in the human body and must be supplemented from outside. Methionine combines with ATP in the human body to generate S-adenosine. It can promote the methylation of liver cell membrane phospholipids, reduce intrahepatic bile stasis, and enhance the transsulfurization effect. It is beneficial for liver cells to restore normal physiological functions, promote the disappearance of jaundice and the recovery of liver function. By supplying methyl groups, it promotes the synthesis of choline, promotes liver fat metabolism, protects the liver and detoxifies. |
59-51-8 | 10 | |
| Thiamine chloride |
Vitamin B1 combines with pyrophosphate in the body to form a cocarboxylase, which participates in the oxidative decarboxylation reactions of pyruvate and α-ketoglutarate in glucose metabolism and is essential for glucose metabolism.
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Vitamin B1 combines with pyrophosphate in the body to form a cocarboxylase, which participates in the oxidative decarboxylation reactions of pyruvate and α-ketoglutarate in glucose metabolism and is essential for glucose metabolism. |
59-43-8 | 9 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| LOMEFLOXACIN, ASPARTATE |
Lomeza is a broad-spectrum quinolone antibiotic that has bactericidal effects on Gram-negative bacteria, Gram-positive bacteria and some anaerobic bacteria. It has good antibacterial effects on methicillin-resistant Staphylococcus aureus, ampicillin-resistant influenza bacilli, pipemidic acid-resistant Escherichia coli and bacteria resistant to other drugs. Its antibacterial mechanism is to inhibit the activity of bacterial DNA helicase, thereby inhibiting bacterial DNA transcription and replication, and has bactericidal effects on a variety of Gram-positive and Gram-negative bacteria.
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Lomeza is a broad-spectrum quinolone antibiotic that has bactericidal effects on Gram-negative bacteria, Gram-positive bacteria and some anaerobic bacteria. It has good antibacterial effects on methicillin-resistant Staphylococcus aureus, ampicillin-resistant influenza bacilli, pipemidic acid-resistant Escherichia coli and bacteria resistant to other drugs. Its antibacterial mechanism is to inhibit the activity of bacterial DNA helicase, thereby inhibiting bacterial DNA transcription and replication, and has bactericidal effects on a variety of Gram-positive and Gram-negative bacteria. |
211690-33-4 | 7 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Dexamethasone sodium phosphate |
Adrenal cortex hormone drugs. It has anti-inflammatory, anti-allergic, anti-rheumatic and immunosuppressive effects. 1 Anti-inflammatory effect: reduce and prevent tissue response to inflammation, inhibit the accumulation of inflammatory cells (including macrophages and leukocytes) at the site of inflammation, inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. 2 Immunosuppressive effect: prevent or inhibit cell-mediated immune response, delayed allergic reaction, reduce the number of T lymphocytes, monocytes, eosinophils, reduce the ability of immunoglobulins to bind to cell surface receptors, and inhibit the synthesis and release of interleukins, thereby reducing the transformation of T lymphocytes to lymphoblasts and reducing the expansion of primary immune responses. It also reduces the passage of immune complexes through the basement membrane and can reduce the concentration of complement components and immunoglobulins.
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Adrenal cortex hormone drugs. It has anti-inflammatory, anti-allergic, anti-rheumatic and immunosuppressive effects. 1 Anti-inflammatory effect: reduce and prevent tissue response to inflammation, inhibit the accumulation of inflammatory cells (including macrophages and leukocytes) at the site of inflammation, inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. 2 Immunosuppressive effect: prevent or inhibit cell-mediated immune response, delayed allergic reaction, reduce the number of T lymphocytes, monocytes, eosinophils, reduce the ability of immunoglobulins to bind to cell surface receptors, and inhibit the synthesis and release of interleukins, thereby reducing the transformation of T lymphocytes to lymphoblasts and reducing the expansion of primary immune responses. It also reduces the passage of immune complexes through the basement membrane and can reduce the concentration of complement components and immunoglobulins. |
55203-24-2 | 25 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Furosemide |
Effects on water and electrolyte excretion: Increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc., inhibit the active reabsorption of NaCl by the thick-walled segment of the renal tubular loop, reduce the reabsorption of Na and Cl-, and promote the secretion of K by the distal tubules; Effects on hemodynamics: Inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, dilate blood vessels, reduce renal vascular resistance, increase renal blood flow, reduce pulmonary capillary permeability, reduce the amount of blood returning to the heart, and reduce left ventricular end-diastolic pressure.
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Effects on water and electrolyte excretion: Increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc., inhibit the active reabsorption of NaCl by the thick-walled segment of the renal tubular loop, reduce the reabsorption of Na and Cl-, and promote the secretion of K by the distal tubules; Effects on hemodynamics: Inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, dilate blood vessels, reduce renal vascular resistance, increase renal blood flow, reduce pulmonary capillary permeability, reduce the amount of blood returning to the heart, and reduce left ventricular end-diastolic pressure. |
54-31-9 | 49 |