Netilmicin is a semi-synthetic aminoglycoside broad-spectrum antibiotic. Its mechanism of action is to inhibit the normal protein synthesis of sensitive microorganisms. This product has a wide range of antimicrobial effects, mainly targeting Gram-negative rods and a few Gram-positive bacteria, including Citrobacter, Enterobacter, Escherichia coli, Klebsiella, Morgan Proteus, Pseudomonas aeruginosa, Salmonella, Shigella and Staphylococcus (penicillin- and methicillin-resistant). This product is also active in vitro against some isolated Acinetobacter, Neisseria, indole-positive Proteus, Pseudomonas, and Serratia. Most streptococci and anaerobic microorganisms such as Clostridium are resistant to aminoglycosides.

This product is a coenzyme that participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotide in the body. When energy is needed for absorption, secretion, muscle contraction and biochemical synthesis reactions in the body, adenosine triphosphate is decomposed into adenosine diphosphate and phosphate groups, and energy is released at the same time. It can penetrate the blood-cerebrospinal fluid barrier, improve the stability and reconstruction ability of the membrane structure of nerve cells, and promote the regrowth of neurites.

This product is a first-generation cephalosporin, which has good antibacterial effect on some strains of Gram-positive cocci such as non-penicillinase-producing and penicillinase-producing Staphylococcus aureus, coagulase-negative Staphylococci, group A hemolytic streptococci, Streptococcus pneumoniae and viridans streptococci. Anaerobic Gram-positive bacteria are mostly sensitive to this product, and Bacteroides fragilis is resistant to this product. Methicillin-resistant Staphylococci and Enterococci are resistant to this product. The effect of this product on Gram-positive and Gram-negative bacteria is similar to that of cephalexin. This product has a certain effect on gonococci and is also active against enzyme-producing gonococci; its activity against Haemophilus influenzae is poor.

1. Stimulate the pancreatic beta cells to secrete insulin, the prerequisite is that the pancreatic beta cells still have a certain ability to synthesize and secrete insulin; 2. Inhibit liver glycogenolysis and gluconeogenesis by increasing portal vein insulin levels or directly acting on the liver, and reduce the production and output of glucose in the liver; 3. May increase the sensitivity of extra-pancreatic tissues to insulin and the utilization of sugar (probably mainly through post-receptor effects). The overall effect is to lower fasting blood sugar and postprandial blood sugar.

It can improve the blood supply to the vertebral artery, regulate the function of the vestibular system, inhibit the vomiting center, and have anti-vertigo and antiemetic effects.

This product can improve the blood supply to the vertebral basilar artery, regulate the function of the vestibular system, inhibit the vomiting center, and has anti-vertigo and antiemetic effects.

This product can improve the blood supply to the vertebral basilar artery, regulate the function of the vestibular system, inhibit the vomiting center, and has anti-vertigo and antiemetic effects.

This product can improve the blood supply to the vertebral basilar artery, regulate the function of the vestibular system, inhibit the vomiting center, and has anti-vertigo and antiemetic effects.

This product can improve the blood supply to the vertebral basilar artery, regulate the function of the vestibular system, inhibit the vomiting center, and has anti-vertigo and antiemetic effects.

This product can improve the blood supply to the vertebral basilar artery, regulate the function of the vestibular system, inhibit the vomiting center, and has anti-vertigo and antiemetic effects.