It interferes with the first step of folic acid synthesis. When used in combination with trimethoprim, it can double block the folic acid metabolism of bacteria, enhance the synergistic antibacterial effect, and reduce drug-resistant strains. It has good antibacterial effects on a variety of bacteria.

It selectively inhibits the action of dihydrofolate reductase. When used in combination with sulfamethoxazole, it can double block bacterial folic acid metabolism, enhance synergistic antibacterial effects, and reduce drug-resistant strains. It has good antibacterial effects on a variety of bacteria.

1. Stimulate the pancreatic islet beta cells to secrete insulin, which presupposes that the islet beta cells also have a certain ability to synthesize and secrete insulin. 2. By increasing portal vein insulin levels or acting directly on the liver, inhibiting liver glycogenolysis and gluconeogenesis, the liver produces and outputs less glucose. 3. It may also increase the sensitivity of extrapancreatic tissues to insulin and the utilization of sugar (probably mainly through post-receptor effects). Therefore, the overall effect is to reduce fasting blood sugar and postprandial blood sugar.

Its stereo configuration is similar to that of lysine, and it can competitively inhibit the adsorption of plasminogen on the fibrin network, protecting the fibrin from being degraded by plasmin and achieving a hemostatic effect.

Quaternary ammonium anticholinergics can selectively act on the digestive tract, inhibit gastric juice secretion and regulate gastrointestinal motility. They can quickly relieve spasms, suppress acid, and relieve pain after taking them. They also have a stronger anti-salivation effect than atropine, but have no central anticholinergic activity.