According to literature reports: 1) It has good transport to organelles in nerve cells, promotes the synthesis of nucleic acids and proteins, and is particularly involved in the synthesis of thymine from deoxynucleotides compared to cyanocobalamin; 2) It promotes intra-axonal transport and axonal regeneration, and can normalize the transport of axonal structural proteins in sciatic nerve cells of diabetic rats given streptozotocin. It has an ameliorative effect on neurotoxicity caused by doxorubicin, acrylamide, and vincristine (in mice).

This product is an intravenous alprostadil preparation with lipid microspheres as drug carriers. Due to the encapsulation of lipid microspheres, alprostadil is not easy to be inactivated. In addition, this product has the targeting characteristics of being easily distributed to damaged blood vessels, thereby exerting the effects of this product in dilating blood vessels and inhibiting platelet aggregation.

Improve blood circulation disorders, reduce vascular resistance, increase myocardial oxygen utilization, etc.

Improve blood circulation disorders, reduce vascular resistance, increase myocardial oxygen utilization, etc.

Improve blood circulation disorders, reduce vascular resistance, increase myocardial oxygen utilization, etc.

Participate in human life activities, promote the enhancement of hematopoietic system activities, increase the number of white blood cells, increase blood vessel elasticity, and prevent vascular sclerosis

Participating in human life activities, essential amino acids have the effects of improving blood circulation disorders, reducing vascular resistance, and increasing myocardial oxygen utilization in cardiovascular diseases.

Participate in human life activities, improve blood circulation disorders, reduce vascular resistance, increase myocardial oxygen utilization, etc.

Essential amino acids participate in human life activities, improve blood circulation disorders, reduce vascular resistance, increase myocardial oxygen utilization, promote the enhancement of hematopoietic system activities, increase the number of white blood cells, and increase vascular elasticity and prevent vascular sclerosis.

This product is a monoquaternary ammonium steroid medium-acting non-depolarizing muscle relaxant, similar in structure to pancuronium bromide, which blocks the conduction between nerve endings and striated muscles by competing with acetylcholine for nicotinic receptors located at the motor end plate of striated muscles. Unlike depolarizing neuromuscular blocking drugs, such as succinylcholine, this product does not cause muscle fiber bundles to tremble. Intravenous injection of 0.08-0.1mg/kg is effective within 1 minute, reaching a peak in 3-5 minutes, and maintaining for 30-90 minutes. The muscle relaxant effect is 3 times stronger than that of tubocurarine chloride; it has no vagus nerve blocking effect. Since vecuronium bromide does not cause an increase in heart rate, it is suitable for patients with myocardial ischemia and heart disease, but the use of vagus nerve excitants and β-receptor blockers is prone to bradycardia. This product has a weak histamine release effect, and there are also bronchospasm and allergic reactions, but they are rare.

Has analgesic effect