It is a penicillin antibiotic. When combined with sulbactam sodium, it is not only protected from enzymatic hydrolysis, but also has a wider antibacterial spectrum. It has good antibacterial activity against enzyme-producing strains of Staphylococcus aureus, Acinetobacter spp. and Bacteroides fragilis.

It is a semi-synthetic β-lactamase inhibitor, which has strong antibacterial activity against Neisseria gonorrhoeae, Neisseria meningitidis and Acinetobacter calcoaceticus, and has strong irreversible competitive inhibition against β-lactamase produced by Staphylococcus aureus and most Gram-negative bacteria. It can be combined with ampicillin to expand the antibacterial spectrum.

Penicillin antibiotics, when combined with sulbactam sodium, are not only protected from enzymatic hydrolysis, but also have a broader antibacterial spectrum, with good antibacterial activity against enzyme-producing strains of Staphylococcus aureus, Acinetobacter spp., and Bacteroides fragilis.

Semi-synthetic β-lactamase inhibitor, with strong antibacterial activity against Neisseria gonorrhoeae, Neisseria meningitidis and Acinetobacter calcoaceticus, and strong irreversible competitive inhibition against β-lactamase produced by Staphylococcus aureus and most Gram-negative bacteria. It can be combined with ampicillin sodium to expand the antibacterial spectrum.

Penicillin antibiotics, when used in combination with sulbactam sodium, can expand the antibacterial spectrum and have good antibacterial activity against enzyme-producing strains of Staphylococci, Streptococci, Pneumococci, Enterococci, Haemophilus influenzae, Moraxella catarrhalis, Escherichia coli, etc.

A semi-synthetic β-lactamase inhibitor that has a strong and irreversible competitive inhibitory effect on β-lactamase produced by Staphylococcus aureus and most Gram-negative bacteria, protecting ampicillin from enzymatic hydrolysis and enhancing its antibacterial effect.

Second-generation cephalosporin antibiotics, which are rapidly hydrolyzed into cefuroxime after entering the body. They have strong antibacterial effects on most Gram-positive cocci, good effects on diphtheria Corynebacterium and Gram-positive anaerobic bacteria, strong activity against Escherichia coli, Proteus mirabilis, Klebsiella pneumoniae and Haemophilus influenzae, sensitive to Salmonella typhi, Shigella, Neisseria gonorrhoeae and Neisseria meningitidis, poor effects on Bacteroides fragilis, and resistance to Serratia, Alcaligenes, Acinetobacter and Pseudomonas aeruginosa. The mechanism of action is to bind to penicillin-binding proteins (PBPs) on the bacterial cell membrane, inhibit the synthesis of bacterial septum and cell wall, affect cell division and growth, and ultimately lead to bacterial lysis and death.

It has good antibacterial effect on Escherichia coli, Klebsiella, Proteus, Salmonella typhi, Shigella, Citrobacter and other Enterobacteriaceae and Pseudomonas aeruginosa. Haemophilus influenzae, Neisseria gonorrhoeae and Neisseria meningitidis are highly sensitive to it. It also has good effect on various groups of streptococci and pneumococci, and only has moderate effect on Staphylococcus aureus (methicillin-sensitive strain). It mainly inhibits the synthesis of bacterial cell wall.

It has a weak antibacterial effect itself and is a competitive, irreversible beta-lactamase inhibitor. When used in combination with cefoperazone, it can increase the ability of cefoperazone to resist degradation by various beta-lactamases and produce a significant synergistic effect on cefoperazone.