After passing through the blood-brain-spinal fluid barrier, it decomposes into glutamate and g-aminobutyric acid (GABA): glutamate is involved in information transmission in the central nervous system; g-aminobutyric acid can antagonize the excitotoxic effects of glutamate, improve nerve cell metabolism, maintain nerve stress resistance, reduce the effects of blood ammonia, and improve brain function.

It inhibits the synthesis of bacterial cell walls by binding to bacterial penicillin-binding proteins (PBPs) and has a bactericidal effect. It has the characteristics of broad spectrum and low toxicity, but it is easily hydrolyzed by β-lactamase produced by bacteria and produces drug resistance.

It is a β-lactamase inhibitor that can inhibit Ⅱ-Ⅴ type β-lactamases. It is a competitive and irreversible inhibitor. When used in combination with piperacillin, it enhances the antibacterial activity of piperacillin, expands the antibacterial spectrum, and enhances its antibacterial activity against piperacillin-resistant enzyme-producing bacteria.

Piperacillin is a broad-spectrum penicillin antibiotic that kills bacteria by interfering with the synthesis of bacterial cell walls. It is mainly used to treat infections caused by Pseudomonas aeruginosa and various Gram-negative bacilli, but it is easily hydrolyzed by β-lactamase produced by bacteria and produces drug resistance. Sulbactam can prevent drug-resistant bacteria from destroying penicillins and cephalosporins, and has a significant synergistic effect with sulbactam.

Sulbactam has no antibacterial activity against other bacteria except Neisseria and Acinetobacter, but it has an irreversible inhibitory effect on most important β-lactamases produced by β-lactam antibiotic-resistant strains. Sulbactam can prevent the destruction of penicillins and cephalosporins by resistant bacteria and has a significant synergistic effect with penicillins and cephalosporins.

This product is a first-generation cephalosporin with a broad antibacterial spectrum and strong activity against Gram-positive bacteria. Penicillinase-producing and non-penicillinase-producing Staphylococcus aureus, coagulase-negative Staphylococcus, Streptococcus pyogenes, Streptococcus pneumoniae, Group B hemolytic Streptococcus, Streptococcus viridans, Staphylococcus epidermidis, Corynebacterium diphtheriae, and Bacillus anthracis are all quite sensitive to this product. Enterococci, methicillin-resistant Staphylococcus, Listeria, and Nocardia are resistant. Haemophilus influenzae, Neisseria meningitidis, Moraxella catarrhalis, and Neisseria gonorrhoeae are highly sensitive to this product. Some strains of Escherichia coli, Klebsiella, Salmonella, Shigella, and Proteus are moderately sensitive to this product, while most of the other Gram-negative rods are resistant. Gram-positive anaerobic bacteria are sensitive to this product, and Bacteroides fragilis is resistant to this product. This product mainly inhibits the synthesis of bacterial cell walls.

Dextrorotatory fructose 1,6-diphosphate (FDP) is an intermediate product of glycolysis. It regulates several enzymatic reactions in cells by activating phosphofructokinase, pyruvate kinase and lactate dehydrogenase. FDP can act on cell membranes, promote the cell's uptake of circulating potassium and stimulate the production of high-energy phosphate and 2,3-diphosphoglycerol in cells. In addition, FDP can reduce hemolysis of erythrocytes caused by mechanical trauma and inhibit the production of oxygen free radicals caused by chemical stimulation. In COPD patients, FDP is beneficial for treatment, especially for strengthening respiratory intensity, which is manifested as an increase in PImax, PaO2 and tolerance.