This drug is an immune enhancement drug. It is a polynucleotide peptide (molecular weight less than 5000) extracted from healthy human blood or animal spleen. It is not easily destroyed by RNAse, DNAse and trypsin. This drug is an important factor in cellular immune response. It has no antigenicity and is not an antibody fragment, but it can transmit the cellular immune reactivity of multiple antigens, including bacteria, fungi, viruses, tissue compatibility antigens and tumor-specific antigens, etc. It can enhance the immune reactivity of receptor cells, promote the chemotaxis of macrophages, increase delayed cell response, and improve the body's ability to kill tumors.

By inhibiting cyclooxygenase, it selectively inhibits the synthesis of prostaglandins in the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating to achieve an antipyretic effect. The intensity of its antipyretic effect is similar to that of aspirin. By inhibiting the synthesis and release of prostaglandins, it increases the pain threshold and has an analgesic effect. It is a peripheral analgesic, weaker than aspirin, and is only effective for mild to moderate pain.

It is necessary for the formation of antibodies and collagen, tissue repair (including certain redox reactions), metabolism of phenylalanine, tyrosine, and folic acid, utilization of iron and carbohydrates, synthesis of fats and proteins, maintenance of immune function, hydroxylation and serotonin, maintaining vascular integrity, and promoting non-heme iron absorption.

Methylcobalamin is an endogenous coenzyme B12. It plays an important role as a coenzyme of methionine synthase in the transmethylation reaction of methionine synthesis from homocysteine. It is easily transferred to the organelles of nerve cells, thereby promoting the synthesis of nucleic acids and proteins. It is easier to transfer into the organelles of nerve cells than cyanocobalamin. In experiments on brain cells and spinal cord nerve cells of mice, it participates in the process of synthesizing thymidine from deoxyuridine, promotes the utilization of folic acid and the metabolism of nucleic acids. It can promote the synthesis of nucleic acids and proteins in rats better than cobalamin. It promotes intra-axonal transport and axonal regeneration.

It has multiple pharmacological effects such as anti-inflammatory, anti-allergic and immunosuppressive, including reducing and preventing tissue response to inflammation, preventing or inhibiting cell-mediated immune response, delayed allergic reaction, and reducing the expansion of primary immune response. At the same time, it can counteract the stimulation of bacterial endotoxins on the body, reduce cell damage and play a role in protecting the body.

It has multiple pharmacological effects such as anti-inflammatory, anti-allergic and immunosuppressive. 1 Anti-inflammatory effect: Glucocorticoids reduce and prevent tissue response to inflammation, thereby reducing the manifestation of inflammation. 2 Immunosuppressive effect: prevent or inhibit cell-mediated immune response, delayed allergic reaction, and reduce the expansion of primary immune response. 3 Anti-toxic and anti-shock effect: Glucocorticoids can counteract the stimulation of bacterial endotoxins on the body, reduce cell damage, and play a role in protecting the body.