It can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

This product can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

This product can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

This product can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

This product can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

It can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

This product can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

This product can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

This product can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

This product can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

This product is an antiplatelet drug that can inhibit the release reaction of platelets (such as the release caused by adrenaline, collagen, thrombin, etc.) and inhibit the release of endogenous ADP, 5-HT, etc. Its mechanism of action is to acetylate the cyclooxygenase (i.e., prostaglandin synthase) of platelets, inhibit the formation of cyclic peroxides, reduce the formation of thromboxane A2 (TXA2), thereby inhibiting platelet aggregation and reducing thrombus formation. This product has strong anti-inflammatory and anti-rheumatic effects, and mild and effective antipyretic and analgesic effects. Its mechanism is the result of inhibiting the synthesis of prostaglandins.

1. Effects on water and electrolyte excretion. ① Diuretic effect, increased excretion of urinary sodium, potassium, chloride, phosphorus and magnesium ions, and reduced excretion of urinary calcium. The mechanism of action of this type of drug is mainly to inhibit the reabsorption of sodium chloride in the distal tubule anterior segment and the proximal tubule (less severe), thereby increasing the Na-K exchange in the distal tubule and the collecting duct, and increasing K secretion. Its mechanism of action is not yet fully understood. This type of drug can inhibit carbonic anhydrase activity to varying degrees, so it can explain its effect on the proximal tubule. This type of drug can also inhibit phosphodiesterase activity, reduce the tubular uptake of fatty acids and mitochondrial oxygen consumption, thereby inhibiting the active reabsorption of Na and Cl- by the tubule. ② Antihypertensive effect. In addition to the diuretic and sodium excretion effects, there may be extrarenal mechanisms involved in antihypertensive treatment, which may be to increase the excretion of Na in the gastrointestinal tract. 2. Effects on renal hemodynamics and glomerular filtration function. Due to the reduced reabsorption of water and Na by the renal tubules, the increased pressure within the renal tubules, and the increased water and Na flowing through the distal convoluted tubules, the macula densa is stimulated to increase the secretion of renin and angiotensin in the kidney through the tubular-glomerular reflex, causing renal vasoconstriction, a decrease in renal blood flow, contraction of the glomerular afferent and efferent arterioles, and a decrease in the glomerular filtration rate.

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