It has a direct relaxant effect on the smooth muscles of the respiratory tract. Its mechanism of action includes inhibiting phosphodiesterase to increase the intracellular cAMP content, producing a bronchial dilation effect through the release of endogenous adrenaline and norepinephrine, acting as a purine receptor blocker to counteract the contractile effect of adenine on the respiratory tract, and enhancing the contractility of the diaphragm to improve respiratory function.

It has a direct relaxing effect on the smooth muscles of the respiratory tract. Its mechanism of action includes inhibiting phosphodiesterase, increasing the intracellular cAMP content; partly due to the release of endogenous adrenaline and norepinephrine; as a purine receptor blocker, it can counteract the contraction effect of adenine on the respiratory tract; enhance the contractility of the diaphragm and improve respiratory function.

It has a direct relaxant effect on the smooth muscles of the respiratory tract, and increases the intracellular cAMP content by inhibiting phosphodiesterase; this is partly due to the release of endogenous adrenaline and norepinephrine; as a purine receptor blocker, it can counteract the contractile effect of adenine and other substances on the respiratory tract; it can enhance the contractility of the diaphragm and improve respiratory function.

Nicotinic acid is converted into nicotinamide in the body, and then combined with riboadenin to form nicotinamide adenine dinucleotide (coenzyme I) and nicotinamide adenine dinucleotide phosphate (coenzyme II), which are necessary for lipid amino acid, protein, purine metabolism, tissue respiration oxidation and glycogenolysis. Nicotinic acid can reduce the utilization of coenzyme A; by inhibiting the synthesis of very low density lipoprotein (VLDL), it affects the transport of cholesterol in the blood. Large doses can reduce serum cholesterol and triglyceride concentrations. Nicotinic acid has a peripheral vasodilation effect.

Nicotinic acid is converted into nicotinamide in the body, and then combined with riboadenin to form nicotinamide adenine dinucleotide (coenzyme I) and nicotinamide adenine dinucleotide phosphate (coenzyme II), which are necessary for lipid amino acid, protein, purine metabolism, tissue respiration oxidation and glycogenolysis. Nicotinic acid can reduce the utilization of coenzyme A; by inhibiting the synthesis of very low density lipoprotein (VLDL), it affects the transport of cholesterol in the blood. Large doses can reduce serum cholesterol and triglyceride concentrations. Nicotinic acid has a peripheral vasodilation effect.