It has a strong antibacterial effect on most anaerobic bacteria, and the antibacterial spectrum includes Bacteroides fragilis and other Bacteroides, Fusobacterium, Clostridium perfringens, Eubacterium, Veillonella, Peptococcus and Peptostreptococcus. The bactericidal concentration is slightly higher than the inhibitory concentration. In addition, it also has a strong killing effect on amoeba and Trichomonas. The bactericidal mechanism is that nitro is reduced to a cytotoxin, which acts on the DNA metabolism process of bacteria and promotes cell death. The mechanism against amoeba is to inhibit its redox reaction, causing the nitrogen chain of the protozoa to break.

It is a long-acting tricyclic antihistamine that can relieve nasal or non-nasal symptoms of seasonal allergic rhinitis by selectively antagonizing peripheral H1 receptors.

It is an adrenaline-mimetic drug that can constrict the blood vessels of the nasal mucosa and relieve symptoms of nasal congestion and runny nose.

Inhibits cyclooxygenase activity, thereby blocking the conversion of arachidonic acid to prostaglandins. At the same time, it promotes the combination of arachidonic acid and triglycerides, reduces the concentration of free arachidonic acid in cells, and indirectly inhibits the synthesis of leukotrienes. The inhibitory effect on prostaglandin synthesis is stronger than aspirin and indomethacin.

This product is a broad-spectrum semi-synthetic cephalosporin antibiotic. Its activity against penicillinase-producing Staphylococcus aureus, group A hemolytic streptococci, viridans streptococci and Staphylococcus epidermidis is the same as that of cefadroxil, and its antibacterial effect against non-enzyme-producing Staphylococcus aureus and pneumococci is 2 to 4 times stronger than that of cefadroxil. Its activity against Gram-negative bacilli, including Escherichia coli and Klebsiella pneumoniae, is stronger than that of cefadroxil, and is similar to that of cefadroxil. Its activity against Proteus mirabilis, Salmonella and Shigella is stronger than that of cefadroxil. 2.9 to 8 mg/L of this product can inhibit all Haemophilus influenzae, including strains resistant to ampicillin. Moraxella catarrhalis and Neisseria gonorrhoeae are very sensitive to this product. Indole-positive Proteus, Serratia, Acinetobacter and Pseudomonas aeruginosa are all resistant to this product. The mechanism of action of this product is to inhibit the synthesis of bacterial cell walls.

Ganciclovir is a guanine antiviral drug. It is a homologue of acyclovir but has a stronger effect. It has a strong inhibitory effect on cytomegalovirus in AIDS patients. After entering the cell, it is rapidly phosphorylated to form a monophosphate compound, and then converted into a triphosphate compound by the action of cellular kinase. The phosphorylation process is faster in cells infected with cytomegalovirus. Its triphosphate can competitively inhibit DNA polymerase and be incorporated into the DNA of viruses and host cells, thereby inhibiting DNA synthesis. The inhibitory effect on viral DNA polymerase is stronger than that on host cell DNA polymerase.