It reduces peripheral vascular resistance, increases cardiac output, reduces venous pressure, activates fibrinolysis, reduces fibrinogen levels, prevents platelet aggregation, promotes the synthesis of pyrimidine nucleosides (NAD and NADP), increases the concentration of adenine nucleosides (AMP, ADP and ATP), and finally hinders lipolysis, reducing elevated cholesterol and triglyceride levels

This product is a H2 receptor blocker of the guanyl thiazole class, which has the characteristics of high affinity for H2 receptors, and has a significant inhibitory effect on gastric acid secretion, and has an inhibitory effect on basal secretion and the increase of gastric acid and pepsin caused by various stimuli. This product does not change the gastric emptying rate, does not interfere with pancreatic function, and has no adverse effects on the cardiovascular system and kidney function. It is different from cimetidine, but similar to ranitidine, that is, long-term high-dose treatment does not cause adverse reactions of androgen antagonism such as male breast development, impotence, lack of sexual desire, and female breast pain and galactorrhea. It has no teratogenic, carcinogenic, drug enzyme inhibitory and androgen inhibitory effects.

This product is an amide local anesthetic, which has a biphasic effect of excitation and inhibition on the central nervous system; at low doses, analgesia and drowsiness occur, and the pain threshold is increased; at high doses, the effect or toxicity is enhanced; at sub-toxic blood concentrations, it has an anticonvulsant effect; high blood concentrations may induce convulsions. Low doses can promote the outflow of K+ in myocardial cells, reduce myocardial automaticity, and have an anti-ventricular arrhythmia effect; therapeutic doses have no significant effect on myocardial electrical activity, atrioventricular conduction, and myocardial contraction, but high blood concentrations may lead to a slowing of cardiac conduction velocity, atrioventricular conduction block, inhibition of myocardial contractility, and a reduction in cardiac output.

It has a peripheral anti-M cholinergic receptor effect, can relieve smooth muscle spasms caused by acetylcholine, can also relieve microvascular spasms, and improve microcirculation. It has a relaxing effect on gastrointestinal smooth muscle and inhibits its peristalsis. Its effect is slightly weaker than atropine, and its effect of inhibiting digestive gland secretion is 1/10 of atropine. Its effect of inhibiting salivary gland secretion and dilating pupils is weaker, which is 1/20 to 1/10 of atropine. Because it is not easy to pass through the blood-cerebrospinal fluid barrier, its central effect is also weaker than atropine.

It has the effects of anti-platelet aggregation, dilating arterioles, improving microcirculation, activating blood circulation and removing blood stasis, and has the effect of disaggregating aggregated platelets