This product is an analog of 2'-deoxyguanine nucleotides that can inhibit the replication of herpes viruses. Its mechanism of action is: ganciclovir is first phosphorylated into monophosphate by a protein kinase homolog encoded by cytomegalovirus (CMV) (UL97 gene), and then further phosphorylated into diphosphate and triphosphate by cellular kinases. In CMV-infected cells, the amount of triphosphate is 100 times higher than that in non-infected cells, indicating that this product can be preferentially phosphorylated in infected cells. Once ganciclovir forms triphosphate, it can persist for several days in CMV-infected cells. The triphosphate of ganciclovir can inhibit viral DNA synthesis in the following ways: 1) competitively inhibiting viral DNA polymerase; 2) incorporating into the DNA of viruses and host cells, resulting in the termination of viral DNA extension. Ganciclovir has a stronger effect on viral DNA polymerase than on host polymerase.

A coenzyme for acetylation reactions in the body. It participates in acetylation reactions in the body and plays an important role in the metabolism of sugar, fat and protein, such as the tricarboxylic acid cycle, liver glycogen accumulation, acetylcholine synthesis, lowering cholesterol, regulating blood lipid content and synthesizing steroid substances, which are all closely related to this product.

A coenzyme for acetylation reactions in the body. It participates in acetylation reactions in the body and plays an important role in the metabolism of sugar, fat and protein, such as the tricarboxylic acid cycle, liver glycogen accumulation, acetylcholine synthesis, lowering cholesterol, regulating blood lipid content and synthesizing steroid substances, which are all closely related to this product.

It can prompt the body to increase the plasma concentration of ACTH and cortisone, promote the blood vessel wall to increase the secretion of PGF2alpha, scavenge free radicals in the body, play an anti-inflammatory and anti-exudative role, increase venous tension, accelerate venous blood flow, promote lymphatic return, improve blood circulation and microcirculation, and protect blood vessel walls.

It can prompt the body to increase the plasma concentration of ACTH and cortisone, promote the blood vessel wall to increase the secretion of PGF2alpha, scavenge free radicals in the body, play an anti-inflammatory and anti-exudative role, increase venous tension, accelerate venous blood flow, promote lymphatic return, improve blood circulation and microcirculation, and protect blood vessel walls.

It prompts the body to increase the plasma concentration of ACTH and cortisone, promotes the blood vessel wall to increase the secretion of PGF2alpha, removes free radicals in the body, plays an anti-inflammatory and anti-exudative role, increases venous tension, accelerates venous blood flow, promotes lymphatic return, improves blood circulation and microcirculation, and protects blood vessel walls.

It prompts the body to increase the plasma concentration of ACTH and cortisone, promotes the blood vessel wall to increase the secretion of PGF2alpha, removes free radicals in the body, plays an anti-inflammatory and anti-exudative role, increases venous tension, accelerates venous blood flow, promotes lymphatic return, improves blood circulation and microcirculation, and protects blood vessel walls.