Vitamin B1 combines with adenosine triphosphate to form vitamin B1 pyrophosphate, which is a necessary coenzyme for carbohydrate metabolism. Vitamin B1 can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, and the conduction of nerve impulses is impaired, affecting gastrointestinal and myocardial functions.

The caffeine in this product can increase the intracellular cyclic adenosine monophosphate (cAMP) content. Small doses can enhance the excitation process of the cerebral cortex, invigorate the spirit, and relieve fatigue. Large doses can excite the medulla oblongata center. Vitamin B1 combines with adenosine triphosphate to form vitamin B1 pyrophosphate, which is a coenzyme necessary for carbohydrate metabolism. Vitamin B1 can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, and the hydrolysis of acetylcholine is accelerated, causing nerve impulse conduction disorders and affecting gastrointestinal and myocardial function. Nicotinic acid can be converted into nicotinamide in the body, and then combined with riboadenine to form nicotinamide adenine dinucleotide (coenzyme I) and nicotinamide adenine dinucleotide phosphate (coenzyme II), which are necessary for lipid, amino acid, protein, purine metabolism, tissue respiration oxidation and glycogenolysis. Nicotinic acid can reduce the effect of coenzyme A; it affects the transportation of cholesterol in the blood by inhibiting the synthesis of very low density lipoprotein (VLDL). Nicotine also has a peripheral vasodilation effect. Sodium glycerophosphate is a phosphorus supplement used to meet the body's daily phosphorus needs. Phosphorus is involved in bone formation and in the composition of cell membranes in the form of phospholipids. Phosphorus is also related to the activity of many metabolic enzymes and plays a vital role in energy metabolism.

Sodium glycerophosphate is a phosphorus supplement used to meet the body's daily phosphorus needs. Phosphorus is involved in bone formation and in the composition of cell membranes in the form of phospholipids. Phosphorus is also related to the activity of many metabolic enzymes and plays a vital role in energy metabolism.

Caffeine can increase the content of cyclic adenosine monophosphate (cAMP) in cells. Small doses can enhance the excitation process of the cerebral cortex, invigorate the spirit, and relieve fatigue. Large doses can excite the medulla oblongata center.

Vitamin B1 combines with adenosine triphosphate to form vitamin B1 pyrophosphate, which is a necessary coenzyme for carbohydrate metabolism. Vitamin B1 can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, and the conduction of nerve impulses is impaired, affecting gastrointestinal and myocardial functions.

Nicotinic acid can be converted into nicotinamide in the body, and then combined with riboadenin to form nicotinamide adenine dinucleotide (coenzyme I) and nicotinamide adenine dinucleotide phosphate (coenzyme II), which are essential for lipid, amino acid, protein, purine metabolism, tissue respiration oxidation and glycogenolysis. Nicotinic acid can reduce the effect of coenzyme A; it affects the transport of cholesterol in the blood by inhibiting the synthesis of very low density lipoprotein (VLDL). Nicotine also has the effect of peripheral vasodilation.

Vitamin B1 is an important component of coenzymes required for sugar metabolism

Vitamin B2 is an important coenzyme component required for tissue respiration. Nicotinamide is a component of coenzymes I and II, and is necessary for lipid metabolism and oxidation of tissue respiration.

Vitamin B6 is a cofactor for many enzymes and is involved in the metabolism of amino acids and fats.

Nicotinamide is a component of coenzymes I and II, essential for lipid metabolism and oxidation of tissue respiration

Calcium pantothenate is a component of coenzyme A and is involved in the metabolism of sugar, fat, and protein.

This product belongs to the macrolide antibiotics, which is the dodecyl sulfate of erythromycin propionate. It has antibacterial activity against Staphylococcus, various groups of streptococci and Gram-positive bacilli. Neisseria, Haemophilus influenzae, Bordetella pertussis, etc. may also be sensitive to this product. This product also has antibacterial activity against various anaerobic bacteria except Bacteroides fragilis and Fusobacterium; it also has inhibitory effects on Legionella, Campylobacter fetus, some spirochetes, Mycoplasma pneumoniae, Rickettsia and Chlamydia. This product is an antibacterial agent, but it also has a bactericidal effect on some bacteria at high concentrations. This product can pass through the bacterial cell membrane and reversibly bind to the 50S subunit of the bacterial ribosome near the donor site (P site), blocking the transfer RNA (t-RNA) binding to the P site, and also blocking the displacement of the polypeptide chain from the acceptor site (A site) to the P site, thereby inhibiting bacterial protein synthesis. This product is only effective against bacteria with active division.

Iron is a component of hemoglobin in red blood cells. When iron is deficient, the amount of hemoglobin synthesized by red blood cells decreases, causing the red blood cells to become smaller and their oxygen-carrying capacity to decrease, leading to iron deficiency anemia. Oral administration of this product can supplement iron and correct iron deficiency anemia.

Iron is a component of hemoglobin in red blood cells. When iron is deficient, the amount of hemoglobin synthesized by red blood cells decreases, causing the red blood cells to become smaller and their oxygen-carrying capacity to decrease, resulting in iron-deficiency anemia. Oral administration of this product can supplement iron and correct iron-deficiency anemia.

Iron is a component of hemoglobin in red blood cells. When iron is deficient, the amount of hemoglobin synthesized by red blood cells decreases, causing the red blood cells to become smaller and their oxygen-carrying capacity to decrease, resulting in iron-deficiency anemia. Oral administration of this product can supplement iron and correct iron-deficiency anemia.

Iron is a component of hemoglobin in red blood cells. When iron is deficient, the amount of hemoglobin synthesized by red blood cells decreases, causing the red blood cells to become smaller and their oxygen-carrying capacity to decrease, resulting in iron-deficiency anemia. Oral administration of this product can supplement iron and correct iron-deficiency anemia.

Can promote granulation tissue and mucosal regeneration

It can inhibit gastric juice secretion and relieve pain caused by smooth muscle spasm

Can neutralize excess stomach acid to relieve symptoms such as stomach pain and heartburn

Can neutralize excess stomach acid to relieve symptoms such as stomach pain and heartburn