It promotes epidermal cell renewal, regulates epidermal cell proliferation and differentiation, makes stratum corneum cells loose and easy to fall off, helps remove acne, and inhibits the formation of new acne.

This product is a nucleoside antiviral drug, which has an inhibitory effect on type I and type II herpes simplex viruses in vitro. In virus-infected cells, viral thymidine deoxynucleoside kinase phosphorylates this product into penciclovir monophosphate, and then cellular kinase converts penciclovir monophosphate into penciclovir triphosphate. In vitro experiments have shown that penciclovir triphosphate and deoxyguanosine triphosphate competitively inhibit herpes simplex virus polymerase, thereby selectively inhibiting the synthesis and inhibition of herpes simplex virus DNA. The generation of herpes simplex virus mutants resistant to this product is due to changes in the properties of viral thymidine deoxynucleoside kinase or DNA polymerase. The most common acyclovir-resistant virus mutants lack thymidine kinase, and they are also resistant to this product.

This product is a glucocorticoid drug, which has anti-inflammatory, anti-allergic, antipruritic and exudation-reducing effects when used externally.

This product is a fluoroquinolone with a broad-spectrum antibacterial effect, especially high antibacterial activity against aerobic Gram-negative bacteria. It has good antibacterial activity in vitro against the following bacteria: most bacteria of the Enterobacteriaceae family, including Citrobacter, Enterobacter cloacae, Enterobacter aerogenes and other Enterobacteriaceae, Escherichia coli, Klebsiella, Proteus, Salmonella, Shigella, Vibrio, Yersinia, etc. Fluoroquinolones often have antibacterial activity against multi-drug resistant bacteria. It has high antibacterial activity against penicillin-resistant gonococci, enzyme-producing influenza bacilli and Moraxella. Fluoroquinolones have poor antibacterial effect against anaerobic bacteria. In recent years, the resistance of bacteria to fluoroquinolones has increased significantly, especially Escherichia coli, with a resistance rate of more than 50%. The resistance of Staphylococcus aureus and Pseudomonas aeruginosa has also increased, and different varieties of various fluoroquinolones are cross-resistant. Fluoroquinolones are fungicides. It is generally believed that quinolones act on the A subunit of DNA helicase in bacterial cells, inhibiting DNA synthesis and replication and causing bacterial death.

Terbinafine hydrochloride is a broad-spectrum antifungal drug of the allylamine class. It can specifically interfere with the early biosynthesis of fungal sterols, selectively inhibit the activity of fungal squalene epoxidase, and hinder the squalene epoxidation reaction in the process of fungal cell membrane formation, thereby achieving the effect of killing or inhibiting fungi.