By protecting the energy metabolism of cells under hypoxia or ischemia, preventing the decrease of intracellular ATP levels, it ensures the normal function of ion pumps and the normal operation of transmembrane sodium-potassium flow, and maintains the stability of the intracellular environment. It helps maintain the energy metabolism of the heart and nerve sensory organs under ischemia and hypoxia, reduces intracellular acidosis and changes in transmembrane ion flow caused by ischemia, reduces the movement and infiltration of multinuclear neutrophils during ischemia and myocardial reperfusion, and reduces the scope of experimental infarction. While producing this effect, it has no significant effect on hemodynamics. It increases coronary blood flow reserve, delays the occurrence of exercise-induced ischemia, limits rapid fluctuations in blood pressure without significant changes in heart rate, significantly reduces the frequency of angina attacks, and significantly reduces the use of glyceryl trinitrate.

This product is an H2 receptor inhibitor, which has the effect of inhibiting gastric acid secretion. It is rapidly absorbed through the gastrointestinal tract after oral administration.

This product is a broad-spectrum antifungal drug, which has antibacterial effects on dermatophytes and Candida. Its mechanism of action is to inhibit the synthesis of bacterial cell membranes.

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