-
Founded in:
2002-12-12 -
Country:
China -
Address:
No. 7, Chuangxin Road, New Industrial Park, High-tech Zone, Xi'an, Shaanxi Province -
Tax NO.:
91610000745007979F -
Registered Funds:
171 million yuan -
Website:
-
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Pancreatin |
Pancreatic enzymes are a mixture of various enzymes, mainly including trypsin, pancreatic amylase and pancreatic lipase, which are more active under neutral or weakly alkaline conditions. In the intestinal fluid, they convert starch into sugar, protein into peptone and fat into glycerol and fatty acids.
More
Pancreatic enzymes are a mixture of various enzymes, mainly including trypsin, pancreatic amylase and pancreatic lipase, which are more active under neutral or weakly alkaline conditions. In the intestinal fluid, they convert starch into sugar, protein into peptone and fat into glycerol and fatty acids. |
50mg | 8049-47-6 | 27 |
| Pig gallbladder ointment |
The pancreatic enzyme in this product is a mixture of various enzymes, mainly including trypsin, pancreatic amylase and pancreatic lipase, etc., which are more active under neutral or weakly alkaline conditions. In the intestinal fluid, it converts starch into sugar, protein into peptone and fat into glycerol and fatty acids. Sodium bicarbonate can enhance the activity of pancreatic enzymes.
More
The pancreatic enzyme in this product is a mixture of various enzymes, mainly including trypsin, pancreatic amylase and pancreatic lipase, etc., which are more active under neutral or weakly alkaline conditions. In the intestinal fluid, it converts starch into sugar, protein into peptone and fat into glycerol and fatty acids. Sodium bicarbonate can enhance the activity of pancreatic enzymes. |
50mg | 0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Diammonium glycyrrhizinate |
It has strong anti-inflammatory, liver cell membrane protection and liver function improvement effects. It can reduce the increase of serum alanine aminotransferase and aspartate aminotransferase caused by carbon tetrachloride, thioacetamide and D-galactosamine, significantly reduce the morphological damage of D-galactosamine to the liver and improve the chronic damage of immune factors to the liver morphology.
More
It has strong anti-inflammatory, liver cell membrane protection and liver function improvement effects. It can reduce the increase of serum alanine aminotransferase and aspartate aminotransferase caused by carbon tetrachloride, thioacetamide and D-galactosamine, significantly reduce the morphological damage of D-galactosamine to the liver and improve the chronic damage of immune factors to the liver morphology. |
79165-06-3 | 35 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Metoprolol tartrate |
This product competitively inhibits the action of catecholamines at the adrenaline beta receptor site, mainly by selectively inhibiting cardiac beta 1 receptors, making them in an inhibited state, but it has a weaker effect on beta 2 receptors. 1 Reduces heart rate and cardiac output at rest and during exercise. 2 Reduces systolic blood pressure during exercise. 3 Inhibits tachycardia caused by isoproterenol. 4 Reduces reflex orthostatic tachycardia. 5 In asthmatic patients, metoprolol tartrate reduces FEV1 (forced expiratory volume in 1 second) and FVC (forced vital capacity) significantly less than non-selective beta blockers (such as propranolol).
More
This product competitively inhibits the action of catecholamines at the adrenaline beta receptor site, mainly by selectively inhibiting cardiac beta 1 receptors, making them in an inhibited state, but it has a weaker effect on beta 2 receptors. 1 Reduces heart rate and cardiac output at rest and during exercise. 2 Reduces systolic blood pressure during exercise. 3 Inhibits tachycardia caused by isoproterenol. 4 Reduces reflex orthostatic tachycardia. 5 In asthmatic patients, metoprolol tartrate reduces FEV1 (forced expiratory volume in 1 second) and FVC (forced vital capacity) significantly less than non-selective beta blockers (such as propranolol). |
56392-17-7 | 51 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Sodium ferulic |
This product is a non-peptide endothelin receptor antagonist, which can antagonize endothelin-induced vasoconstriction, blood pressure increase and vascular smooth muscle cell proliferation, reduce vascular endothelial damage; increase NO synthesis, relax vascular smooth muscle; inhibit platelet aggregation, anti-coagulation, and improve blood rheology characteristics. This product can also inhibit cholesterol synthesis, lower blood lipids, scavenge free radicals, prevent lipid peroxidation damage; affect complement, enhance immune function, and have certain analgesic and antispasmodic effects.
More
This product is a non-peptide endothelin receptor antagonist, which can antagonize endothelin-induced vasoconstriction, blood pressure increase and vascular smooth muscle cell proliferation, reduce vascular endothelial damage; increase NO synthesis, relax vascular smooth muscle; inhibit platelet aggregation, anti-coagulation, and improve blood rheology characteristics. This product can also inhibit cholesterol synthesis, lower blood lipids, scavenge free radicals, prevent lipid peroxidation damage; affect complement, enhance immune function, and have certain analgesic and antispasmodic effects. |
24276-84-4 | 8 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Propyl gallate |
1. It can obviously inhibit the synthesis of thromboxane A2 (TXA2), counteract the platelet aggregation caused by arachidonic acid (AA), and has a stronger and faster antiplatelet aggregation effect than aspirin (ASP). 2. It can enhance the fibrinolytic activity and promote the dissolution of thrombus. 3. It can reduce the specific viscosity of whole blood and plasma, and accelerate the electrophoresis of red blood cells. 4. It can relax the vascular smooth muscle, dilate the artery, and increase the blood flow of the coronary artery and cerebral artery. 5. It can improve the ability to resist hypoxia. 6. It can strongly scavenge free radicals. 7. It can inhibit the inflammatory exudation of capillaries.
More
1. It can obviously inhibit the synthesis of thromboxane A2 (TXA2), counteract the platelet aggregation caused by arachidonic acid (AA), and has a stronger and faster antiplatelet aggregation effect than aspirin (ASP). 2. It can enhance the fibrinolytic activity and promote the dissolution of thrombus. 3. It can reduce the specific viscosity of whole blood and plasma, and accelerate the electrophoresis of red blood cells. 4. It can relax the vascular smooth muscle, dilate the artery, and increase the blood flow of the coronary artery and cerebral artery. 5. It can improve the ability to resist hypoxia. 6. It can strongly scavenge free radicals. 7. It can inhibit the inflammatory exudation of capillaries. |
121-79-9 | 10 |