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Founded in:
1989-10-20 -
Country:
China -
Address:
No. 185, Liaocheng East Road, Gushi County -
Tax NO.:
91411525706758024M -
Registered Funds:
84 million yuan -
Website:
-
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Acetaminophen |
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects
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This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects |
0.3g | 103-90-2 | 68 |
| Starch |
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects.
More
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects. |
9005-25-8 | 79 | |
| Pregelatinized starch (medicinal excipients) |
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects.
More
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects. |
0 | ||
| Polyvinylpyrrolidone |
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects.
More
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects. |
9003-39-8 | 18 | |
| Stearic acid |
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects.
More
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects. |
57-11-4 | 26 | |
| Talc |
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects.
More
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects. |
14807-96-6 | 26 | |
| Potassium sorbate |
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects.
More
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects. |
24634-61-5 | 8 | |
| Hydroxypropyl methyl cellulose |
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects.
More
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects. |
9004-65-3 | 56 | |
| Triacetin |
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects.
More
This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects. |
102-76-1 | 10 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| CALCIUM GLUCONATE MONOHYDRATE |
This product is a calcium supplement. Calcium can maintain the normal excitability of nerves and muscles and promote the secretion of acetylcholine by nerve endings. When serum calcium is low, the excitability of nerves and muscles may increase, resulting in convulsions. When blood calcium is too high, the excitability may decrease, resulting in weakness, etc. Calcium ions can improve the permeability of cell membranes, increase the density of capillaries, reduce exudation, and have an anti-allergic effect. Calcium ions can promote the calcification of bones and teeth. There is a competitive antagonism between high concentrations of calcium ions and magnesium ions, which can be used to rescue magnesium poisoning; calcium ions can react with fluoride to form insoluble calcium fluoride, which can be used to rescue fluoride poisoning.
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This product is a calcium supplement. Calcium can maintain the normal excitability of nerves and muscles and promote the secretion of acetylcholine by nerve endings. When serum calcium is low, the excitability of nerves and muscles may increase, resulting in convulsions. When blood calcium is too high, the excitability may decrease, resulting in weakness, etc. Calcium ions can improve the permeability of cell membranes, increase the density of capillaries, reduce exudation, and have an anti-allergic effect. Calcium ions can promote the calcification of bones and teeth. There is a competitive antagonism between high concentrations of calcium ions and magnesium ions, which can be used to rescue magnesium poisoning; calcium ions can react with fluoride to form insoluble calcium fluoride, which can be used to rescue fluoride poisoning. |
66905-23-5 | 35 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Rifampicin |
Rifampicin is a semi-synthetic broad-spectrum antibacterial drug of the rifamycin class, which has antibacterial activity against a variety of pathogenic microorganisms. The drug has a significant bactericidal effect on Mycobacterium tuberculosis and some non-tuberculous mycobacteria (including Mycobacterium leprae, etc.) both inside and outside the host cells. Rifampicin has a good antibacterial effect on aerobic Gram-positive bacteria, including enzyme-producing strains of Staphylococcus aureus and methicillin-resistant strains, Streptococcus pneumoniae, other Streptococcus, Enterococcus, Listeria, Bacillus anthracis, Clostridium perfringens, Corynebacterium diphtheriae, anaerobic cocci, etc. It also has a high degree of antibacterial activity against aerobic Gram-negative bacteria such as Neisseria meningitidis, Haemophilus influenzae, and Neisseria gonorrhoeae. Rifampicin also has a good effect on Legionella, and has an inhibitory effect on pathogens such as Chlamydia trachomatis, lymphogranuloma venereum, and psittacosis.
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Rifampicin is a semi-synthetic broad-spectrum antibacterial drug of the rifamycin class, which has antibacterial activity against a variety of pathogenic microorganisms. The drug has a significant bactericidal effect on Mycobacterium tuberculosis and some non-tuberculous mycobacteria (including Mycobacterium leprae, etc.) both inside and outside the host cells. Rifampicin has a good antibacterial effect on aerobic Gram-positive bacteria, including enzyme-producing strains of Staphylococcus aureus and methicillin-resistant strains, Streptococcus pneumoniae, other Streptococcus, Enterococcus, Listeria, Bacillus anthracis, Clostridium perfringens, Corynebacterium diphtheriae, anaerobic cocci, etc. It also has a high degree of antibacterial activity against aerobic Gram-negative bacteria such as Neisseria meningitidis, Haemophilus influenzae, and Neisseria gonorrhoeae. Rifampicin also has a good effect on Legionella, and has an inhibitory effect on pathogens such as Chlamydia trachomatis, lymphogranuloma venereum, and psittacosis. |
13292-46-1 | 24 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Nitrendipine |
This product is a dihydropyridine calcium channel blocker that inhibits the transmembrane calcium ion influx of vascular smooth muscle and myocardium. It mainly acts on blood vessels, causing the dilation of systemic blood vessels such as coronary arteries and renal arterioles, producing a hypotensive effect.
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This product is a dihydropyridine calcium channel blocker that inhibits the transmembrane calcium ion influx of vascular smooth muscle and myocardium. It mainly acts on blood vessels, causing the dilation of systemic blood vessels such as coronary arteries and renal arterioles, producing a hypotensive effect. |
39562-70-4 | 23 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Nifedipine |
By blocking the membrane transport of calcium ions in myocardial and vascular smooth muscle, inhibiting the influx of calcium ions into cells, it causes a decrease in myocardial contractility and vasodilation. Animal experiments have shown that by reducing myocardial contractility and peripheral vascular resistance, myocardial oxygen consumption is reduced; by dilating coronary vessels and developing collateral circulation, the oxygen supply to myocardial ischemic sites is increased; by inhibiting the consumption of high-energy phosphate compounds, the ability to resist hypoxia is enhanced.
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By blocking the membrane transport of calcium ions in myocardial and vascular smooth muscle, inhibiting the influx of calcium ions into cells, it causes a decrease in myocardial contractility and vasodilation. Animal experiments have shown that by reducing myocardial contractility and peripheral vascular resistance, myocardial oxygen consumption is reduced; by dilating coronary vessels and developing collateral circulation, the oxygen supply to myocardial ischemic sites is increased; by inhibiting the consumption of high-energy phosphate compounds, the ability to resist hypoxia is enhanced. |
21829-25-4 | 75 |