It is a potent, orally effective, selective angiotensin-II receptor (AT1 subtype) antagonist that can block all AT1 receptor-mediated angiotensin-II effects. The selective antagonism of angiotensin-II receptor (AT1) leads to increased plasma renin and angiotensin-II levels and decreased plasma aldosterone levels. It does not inhibit angiotensin converting enzyme (ACE or kinase II), under the action of which angiotensin-II can be generated, and bradykinin can also be degraded into inactive metabolites. The activity of irbesartan does not require metabolic activation.

It is a thiazide diuretic that affects the renal tubular reabsorption mechanism of electrolytes, directly increases the excretion of sodium and chloride (roughly equal amounts), reduces blood volume, increases plasma renin activity, increases aldosterone secretion, thereby increasing the excretion of potassium and bicarbonate in urine and reducing serum potassium levels. The combined use of irbesartan can reverse the loss of potassium associated with diuretics by blocking the renin-angiotensin-aldosterone system.

Guaifenesin is an expectorant that can stimulate the gastric mucosa to reflexively cause increased secretion of bronchial mucosal glands, reduce the viscosity of sputum, and make sticky sputum easier to cough up.

This product was originally an antiviral drug. Its anti-Parkinson's disease mechanism is mainly to promote the synthesis and release of striatal dopamine, reduce the reuptake of dopamine by nerve cells, and have an anti-acetylcholine effect, thereby improving the symptoms of Parkinson's patients.

Broad-spectrum antiviral drug. It has the effect of inhibiting the growth of multiple viruses such as respiratory syncytial virus, influenza virus, hepatitis A virus, adenovirus, etc. in vitro, and its mechanism is not fully understood. After entering the virus-infected cells, the drug is rapidly phosphorylated, and its product acts as a competitive inhibitor of viral synthase, inhibiting inosine monophosphate dehydrogenase, influenza virus RNA polymerase and mRNA guanosine transferase, thereby causing a decrease in intracellular guanosine triphosphate, impairing viral RNA and protein synthesis, and inhibiting viral replication and transmission. It may also have an immune effect and neutralizing antibody effect on respiratory syncytial virus.