This product is a quinolone antibacterial drug, which has strong antibacterial effect on Gram-negative bacteria, including Escherichia coli, Klebsiella pneumoniae, Proteus, Salmonella typhi, Salmonella paratyphi, Shigella, Enterobacter cloacae, Enterobacter aerogenes, Citrobacter, Serratia marcescens, Pseudomonas aeruginosa, Neisseria meningitidis, Haemophilus influenzae, Moracata, Legionella pneumophila, Neisseria gonorrhoeae, etc. It also has moderate antibacterial effect on Gram-positive cocci such as Staphylococcus and hemolytic Streptococcus. The mechanism of action of this product is to inhibit the bacterial DNA gyrase and thus kill the bacteria.

This product is a brain metabolism improving drug, which is a cyclic derivative of г-aminobutyric acid. It has the effect of resisting brain function damage caused by physical and chemical factors. It can promote the generation of ATP in the brain, promote the synthesis of acetylcholine and enhance the conduction of nerve excitement, and has the effect of promoting brain metabolism. It can resist brain function damage caused by physical and chemical factors. It has an improving effect on retrograde amnesia caused by hypoxia. It can enhance memory and improve learning ability.

It can clear away heat and dampness, promote diuresis and reduce jaundice, detoxify, improve the symptoms and signs of pathological hepatitis, reduce jaundice, and reduce enzymes. Animal in vitro tests have shown that it can inhibit the replication of hepatitis B virus.

1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc., mainly by inhibiting the active reabsorption of NaCl by the thick-walled segment of the renal tubular loop, reducing the osmotic pressure gradient difference of the medullary interstitial fluid, resulting in increased excretion of water, Na+, and Cl-. At the same time, it promotes increased Na+-K+ and Na+-H+ exchange in the distal tubules, and increases the excretion of K+ and H+; inhibits the reabsorption of Ca2+ and Mg2+ by the loop of Henle and increases their excretion. Short-term medication can increase uric acid excretion, and long-term medication can cause hyperuricemia. 2. Effect on hemodynamics: Furosemide can inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, and has a vasodilatory effect. It dilates renal blood vessels, reduces renal vascular resistance, and increases renal blood flow, especially deep renal cortical blood flow, which is of great significance in the diuretic effect. Unlike other diuretics, loop diuretics do not decrease the glomerular filtration rate when increasing renal tubular fluid flow. Furosemide can dilate pulmonary capacitance veins, reduce pulmonary capillary permeability, reduce ventricle return volume, and reduce left ventricular end-diastolic pressure, which is helpful for the treatment of acute left heart failure. It also provides a theoretical basis for the treatment of adult respiratory distress syndrome.