This product is an analog of 2-deoxyguanine nucleotide, which can inhibit the replication of herpes virus. Its mechanism of action is: ganciclovir is first phosphorylated into monophosphate by the protein kinase homologue encoded by cytomegalovirus (CMV) (UL97 gene), and then further phosphorylated into diphosphate and triphosphate by cellular kinase. In CMV-infected cells, the amount of triphosphate is 100 times higher than that in non-infected cells, indicating that this product can be preferentially phosphorylated in infected cells. Once ganciclovir forms triphosphate, it can last for several days in CMV-infected cells. Ganciclovir triphosphate can inhibit viral DNA synthesis in the following ways: 1. Competitive inhibition of viral DNA polymerase; 2. Incorporation into the DNA of viruses and host cells, resulting in the termination of viral DNA extension. Ganciclovir has a stronger effect on viral DNA polymerase than on host polymerase. It has been clinically proven that this product is effective against infections caused by cytomegalovirus (CMV) and herpes simplex virus (HSV).

It inhibits bacterial protein synthesis by hindering the bacterial transpeptidation process. It has antibacterial effects on a variety of Gram-positive aerobic bacteria, Gram-negative aerobic bacteria, anaerobic bacteria, sexually transmitted disease microorganisms and other microorganisms. The mechanism of action is the same as that of erythromycin, mainly binding to the 50S subunit of the bacterial ribosome to inhibit RNA-dependent protein synthesis.

This product is a carbonic anhydrase inhibitor that can inhibit the production of aqueous humor and reduce intraocular pressure. Acetazolamide can inhibit the activity of carbonic anhydrase in the ciliary epithelium, thereby reducing the production of aqueous humor (50% to 60%) and lowering intraocular pressure.

Central muscle relaxants mainly act on the spinal cord and subcortical areas of the brain to produce muscle relaxation effects. They take effect within 1 hour after oral administration and last for 3-4 hours.

Pharmacology: This product is a central muscle relaxant, which mainly acts on the spinal cord and subcortical areas of the brain to produce muscle relaxation. It takes effect within 1 hour after oral administration and lasts for 3-4 hours.

Quinolone antibacterial drugs inhibit bacterial DNA helicase and have a broad-spectrum antibacterial effect. They have strong antibacterial activity against Enterobacter bacteria such as Escherichia coli, Klebsiella, Proteus, Shigella, Salmonella typhi, etc., as well as influenza bacillus and Neisseria. They also have certain antibacterial effects on Staphylococcus aureus and Pseudomonas aeruginosa. They have only mild effects on pneumococci, various groups of streptococci and enterococci.