It has the effect of competitively blocking the binding of histamine to H2 receptors and inhibiting gastric acid secretion. It is a potent H2 receptor blocker, and its gastric acid inhibition effect is 5 to 12 times that of cimetidine on a molar basis.

It participates in amino acid metabolism, synthesis of neurotransmitters, synthesis of collagen and tissue cell interstitial, can reduce the permeability of capillaries, accelerate blood coagulation, stimulate coagulation function, promote iron absorption in the intestine, promote the decrease of blood lipids, increase resistance to infection, participate in detoxification function, and has antihistamine effect and prevents the formation of carcinogens (nitrosamines).

It has an antihistamine effect and can compete with histamine released from tissues for the H1 receptors on effector cells, thereby stopping allergic reactions; it inhibits central nervous system activity and causes sedative and hypnotic effects; it enhances the effects of antitussive drugs; it has anti-vertigo and anti-tremor paralysis effects; it has local anesthetic, antiemetic and anti-M cholinergic effects.

This product is a brain metabolism improving drug, which is a cyclic derivative of g-aminobutyric acid. It has the effect of resisting brain function damage caused by physical and chemical factors. It can promote ATP in the brain, promote acetylcholine synthesis and positively enhance the conduction of nerve excitement, and has the effect of promoting brain metabolism. It can resist brain function damage caused by physical and chemical factors. It has an improving effect on retrograde amnesia caused by hypoxia. It can enhance memory and improve learning ability.

Amikacin sulfate is an aminoglycoside antibiotic that has good effects on most Enterobacteriaceae, Pseudomonas aeruginosa and some other Pseudomonas, Acinetobacter, Alcaligenes, etc. It also has good antibacterial effects on meningococci, gonococci, influenza bacilli, Yersinia, Campylobacter fetus, Mycobacterium tuberculosis and some Mycobacterium. Its antibacterial activity is slightly lower than that of gentamicin. Its most prominent advantage is that it is stable against aminoglycoside inactivating enzymes produced by many intestinal Gram-negative bacilli and will not lose its antibacterial activity due to inactivation of such enzymes. The mechanism of action is to act on the 30S subunit of bacterial ribosomes and inhibit bacterial protein synthesis. Combination with semi-synthetic penicillins or cephalosporins often achieves synergistic antibacterial effects.