It is a broad-spectrum semi-synthetic penicillin, which has a broad-spectrum killing effect on Gram-positive and Gram-negative bacteria. It has a strong antibacterial effect on hemolytic streptococci, pneumococci and non-penicillinase-producing staphylococci, which is similar to or slightly inferior to penicillin. It also has a good antibacterial effect on viridans streptococci, and is better than penicillin on enterococci and Listeria. It has antibacterial effects on Corynebacterium diphtheriae, Bacillus anthracis, Actinomyces, Haemophilus influenzae, Bordetella pertussis and Neisseria.

It is an acid-resistant and enzyme-resistant semi-synthetic penicillin that is effective against penicillin-resistant Staphylococcus aureus. They can enhance the effects of each other and make up for the shortcomings of each when used alone.

Ticarcillin is a broad-spectrum penicillin bactericide that can kill a variety of Gram-positive and Gram-negative bacteria. When combined with clavulanic acid, it restores its sensitivity to β-lactamase-producing bacteria.

Clavulanic acid is an irreversible and highly effective β-lactamase inhibitor. It has little antibacterial effect alone, but by blocking β-lactamase, it enhances the antibacterial activity of ticarcillin, making the combination preparation an antibiotic with broad-spectrum bactericidal effect.

This product is a new type of fluoroquinolone antibacterial drug. It inhibits the activity of bacterial DNA helicase and topoisomerase IV, hinders bacterial DNA replication and achieves antibacterial effect. It is effective against aerobic bacteria, general anaerobes, and anaerobic Gram-positive bacteria and Gram-negative bacteria, and its antibacterial spectrum is the same as that of ceftazidime and gentamicin. It has a strong killing effect on Pseudomonas aeruginosa, Escherichia coli, and Staphylococcus aureus, especially against Pseudomonas aeruginosa in the stationary phase, logarithmic growth phase, and stable phase. It is stronger than ceftazidime and gentamicin, and its killing effect on Staphylococcus aureus in the stationary phase and growth phase is also better than ceftazidime.

It is a penicillin antibiotic. When combined with sulbactam sodium, it is not only protected from enzymatic hydrolysis, but also has a broader antibacterial spectrum. It has good antibacterial activity against enzyme-producing strains of Staphylococcus aureus, Acinetobacter spp., and Bacteroides fragilis.

It is a semi-synthetic β-lactamase inhibitor, which has strong antibacterial activity against Neisseria gonorrhoeae, Neisseria meningitidis and Acinetobacter calcoaceticus, and has strong irreversible competitive inhibition against β-lactamase produced by Staphylococcus aureus and most Gram-negative bacteria. When combined with ampicillin sodium, it expands the antibacterial spectrum.

Sulbenzyl penicillin is a broad-spectrum semi-synthetic penicillin antibiotic. It has antibacterial effects on Enterobacteriaceae such as Escherichia coli, Proteus, Enterobacter, Citrobacter, Salmonella and Shigella, as well as other Gram-negative bacteria such as Pseudomonas aeruginosa, Haemophilus influenzae and Neisseria. This product also has antibacterial activity against hemolytic streptococci, Streptococcus pneumoniae and non-penicillinase-producing Staphylococci. This product also has a certain effect on anaerobic bacteria including Peptostreptococci and Clostridium. The mechanism of action of sulbenzyl penicillin is to exert a bactericidal effect by inhibiting bacterial cell wall synthesis.