Wanbangde Pharmaceutical Group Co., Ltd.

By blocking the membrane transport of calcium ions in myocardial and vascular smooth muscle, inhibiting the influx of calcium ions into cells, it causes a decrease in myocardial contractility and vasodilation. Animal experiments have shown that by reducing myocardial contractility and peripheral vascular resistance, myocardial oxygen consumption is reduced; by dilating coronary vessels and developing collateral circulation, the oxygen supply to myocardial ischemic sites is increased; by inhibiting the consumption of high-energy phosphate compounds, the ability to resist hypoxia is enhanced.

It participates in cellular energy metabolism and protein synthesis in the body, increases the activity of related metabolic enzymes, improves liver function, and promotes the recovery of damaged liver.

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It can significantly inhibit the basal gastric acid secretion during the day and night, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.

Proton pump inhibitor. This product is a fat-soluble weakly alkaline drug that is easily concentrated in an acidic environment. After oral administration, it can be specifically distributed in the secretory tubules of gastric mucosal parietal cells. In a high-acid environment, it is converted into an active form of sulfenamide, which irreversibly combines with the sulfhydryl group of H, K-ATPase (also known as proton pump) in the secretory membrane of parietal cells through disulfide bonds to form a complex of sulfenamide and proton pump, thereby inhibiting the activity of the enzyme and blocking the final step of gastric acid secretion. It has a strong and lasting inhibitory effect on gastric acid secretion caused by various reasons.