This product has a broad-spectrum antibacterial effect and strong antibacterial effect. It has strong antibacterial activity against most Enterobacteriaceae, such as Escherichia coli, Klebsiella, Proteus, Salmonella, Shigella, and Gram-negative bacteria such as Haemophilus influenzae, Legionella pneumophila, and Neisseria gonorrhoeae. It also has antibacterial effects on Gram-positive bacteria such as Staphylococcus aureus, Streptococcus pneumoniae, Streptococcus pyogenes, and Mycoplasma pneumoniae and Chlamydia pneumoniae, but has poor effects on anaerobic bacteria and enterococci. Its mechanism of action is to inhibit the activity of bacterial DNA gyrase, prevent the synthesis and replication of bacterial DNA, and cause bacterial death.

1. It can simultaneously relax the coronary arteries in the normal blood supply area and the ischemic area, antagonize spontaneous or ergonovine-induced coronary artery spasm, increase the delivery of myocardial oxygen to patients with coronary artery spasm, and relieve and prevent coronary artery spasm. 2. It can inhibit myocardial contraction, reduce myocardial metabolism, and reduce myocardial oxygen consumption. 3. It can relax peripheral resistance vessels, reduce peripheral resistance, reduce systolic and diastolic blood pressure, and reduce cardiac afterload. 4. It can delay the sinus node function and atrioventricular conduction of the isolated heart; electrophysiological studies on whole animals and humans have not found that this product has the effect of delaying atrioventricular conduction, prolonging the recovery time of the sinus node, and slowing down the sinus node rate.

Competitively blocks the binding of histamine to H2 receptors, inhibits gastric acid secretion, and is 5 to 12 times more potent than cimetidine (in moles), making it a potent H2 receptor blocker

The specific pharmacological effects are not clearly described from the above information

By inhibiting the reabsorption of cholesterol in the intestine and reducing the secretion of cholesterol into the bile, the saturation of cholesterol in the bile is reduced. Cholesterol stones may be gradually dissolved due to the dispersion of cholesterol and the formation of liquid crystals. The treatment of liver and cholestatic diseases is mainly based on the relative replacement of lipophilic, detergent-like toxic bile acids by hydrophilic, cytoprotective and non-cytotoxic ursodeoxycholic acid, as well as the promotion of hepatocyte secretion and immunomodulation.