It combines with poisons containing hydroxyl or carboxyl groups to form low-toxic or non-toxic compounds that are excreted in the urine. It has the effect of protecting the liver and detoxifying. It can increase the glycogen content of the liver and reduce fat reserves.

After entering the body, glucuronide can combine with poisons containing hydroxyl or carboxyl groups to form low-toxic or non-toxic combinations that are excreted in the urine, which has the effect of protecting the liver and detoxifying. In addition, glucuronic acid can increase the glycogen content of the liver and reduce fat reserves. Vitamin B1 combines with adenosine triphosphate to form vitamin B1 pyrophosphate (thiamine triphosphate, cocarboxylase), which is a coenzyme necessary for carbohydrate metabolism; vitamin B1 can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase increases, the hydrolysis of acetylcholine is accelerated, and the conduction of nerve impulses is impaired, affecting gastrointestinal and myocardial functions. This product is mainly absorbed in the duodenum, metabolized in the liver, and excreted through the kidneys.

It combines with adenosine triphosphate to form vitamin B1 pyrophosphate (thiamine triphosphate, cocarboxylase), which is a coenzyme necessary for carbohydrate metabolism; it can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase increases, the hydrolysis of acetylcholine is accelerated, causing nerve impulse conduction disorders, and affecting gastrointestinal and myocardial function.

Clonidine is an alpha receptor agonist. Clonidine directly stimulates the alpha 2 receptors on the central postsynaptic membrane of the hypothalamus and medulla oblongata, excites inhibitory neurons, reduces the outflow of central sympathetic nerve impulses, and thus inhibits peripheral sympathetic nerve activity. Clonidine also stimulates the alpha 2 receptors on the presynaptic membrane of peripheral sympathetic nerves, enhancing their negative feedback, reducing the release of norepinephrine from peripheral nerve terminals, reducing peripheral vascular and renal vascular resistance, slowing heart rate, and lowering blood pressure. Renal blood flow and glomerular filtration rate remain essentially unchanged. Orthostatic symptoms are mild or less common, and postural hypotension rarely occurs. Clonidine hydrochloride causes a moderate decrease (15%~20%) in supine cardiac output without changing peripheral vascular resistance; a slight decrease in 45° tilt

No specific pharmacological information is provided

This product is a derivative of norephedrine, which selectively stimulates beta receptors and directly dilates coronary arteries, can reduce coronary resistance, increase coronary flow, reduce ventricular volume, reduce ventricular wall tension, improve myocardial oxygen balance, enhance myocardial contractility, and accelerate heart rate. It has little effect on blood pressure.