It has the effects of lowering blood pressure, slowing heart rate and preventing arrhythmia

It has the effects of lowering blood pressure, slowing heart rate and preventing arrhythmia

It has the effects of lowering blood pressure, slowing heart rate and preventing arrhythmia

Not clearly mentioned, may be an auxiliary blood pressure lowering or gastrointestinal protection effect

Has blood pressure lowering effect

Has blood pressure lowering effect

Has diuretic and blood pressure lowering effects

It has a sedative effect, reduces spontaneous activity and works synergistically with sodium pentobarbital

Not explicitly mentioned, may be an auxiliary nutritional support

Not explicitly mentioned, may be an auxiliary nutritional support

Not explicitly mentioned, may be an auxiliary nutritional support

It achieves antibacterial effect by inhibiting the activity of bacterial DNA gyrase (bacterial topoisomerase II) and hindering the replication of bacterial DNA. It has good antibacterial effect on most Enterobacteriaceae, Gram-negative bacteria, some Gram-positive bacteria, Legionella, Mycoplasma, Chlamydia, but has poor effect on anaerobic bacteria and enterococci.

This product is an amide local anesthetic. After blood absorption or intravenous administration, it has obvious biphasic effects of excitation and inhibition on the central nervous system, and there may be no precursor excitation. When the blood concentration is low, analgesia and drowsiness occur, and the pain threshold increases; with increasing doses, the effect or toxicity increases, and it has an anticonvulsant effect at sub-toxic blood concentrations; convulsions may occur when the blood concentration exceeds 5μg·ml-1. At low doses, it can promote the outflow of K in myocardial cells, reduce the autonomy of the myocardium, and have an anti-ventricular arrhythmia effect; at therapeutic doses, it has no obvious effect on the electrical activity of myocardial cells, atrioventricular conduction, and myocardial contraction; further increases in blood concentrations can cause a slowing of the heart conduction velocity, atrioventricular conduction block, inhibition of myocardial contractility, and a decrease in cardiac output.

Nimodipine is a Ca2 channel blocker that inhibits smooth muscle contraction and relieves vasospasm by preventing Ca2 from entering cells. It has a strong effect on cerebral arteries and can penetrate the blood-brain barrier to prevent vasospasm after subarachnoid hemorrhage, protect and promote memory, promote intellectual recovery, selectively dilate cerebral blood vessels, increase cerebral blood flow, and reduce ischemic brain damage.

Non-digitalis cardiotonic agent with positive inotropic effect, can enhance myocardial contractility, improve cardiac pumping function, dilate blood vessels, reduce myocardial oxygen consumption; improve myocardial cell metabolism, protect ischemic and hypoxic myocardium; can improve sinoatrial node P cell function.