Not clearly described

This product is widely used to treat diabetic neuropathy. In vitro tests show that this product can reduce lipid oxidation in nerve tissues. This product may prevent the glycosylation of proteins: and it can inhibit aldose reductase, thereby preventing the conversion of glucose or galactose into sorbitol. Animal experiments suggest that lipoic acid can prevent the development of diabetes, promote the utilization of glucose, and prevent neuropathy caused by hyperglycemia. Lipoic acid is a strong antioxidant in both water-soluble and oil-soluble matrices. Both lipoic acid and its reduced form, dilipoic acid, can play an antioxidant role. They can directly or indirectly promote the regeneration of vitamin C and vitamin E in the body. Studies have shown that lipoic acid can increase the levels of glutathione and coenzyme Q10 in cells. Lipoic acid can chelate certain metal ions (such as copper, manganese, and zinc) to form stable chelates. In animal models, it has been shown that it can protect against arsenic poisoning and reduce liver toxicity after chromium poisoning. In in vitro experiments, it was also found that mercury ions can be chelated from kidney slices.

1. This product is widely used to treat diabetic neuropathy. In vitro tests show that this product can reduce lipid oxidation in nerve tissue, and this product may prevent protein glycosylation: and can inhibit aldose reductase, thereby preventing glucose or galactose from being converted into sorbitol. 2. Animal experiments suggest that lipoic acid can prevent the development of diabetes, promote the utilization of glucose, and prevent neuropathy caused by hyperglycemia. Lipoic acid is a strong antioxidant in both water-soluble and oil-soluble matrices. Both lipoic acid and its reduced form, dilipoic acid, can play an antioxidant role. They can directly or indirectly promote the regeneration of vitamin C and vitamin E in the body. Studies have shown that lipoic acid can increase the levels of glutathione and coenzyme Q10 in cells. 3. Lipoic acid can chelate certain metal ions (such as copper, manganese, and zinc) to form stable chelates. In animal models, it has been shown that it can protect against arsenic poisoning and reduce liver toxicity after chromium poisoning. In in vitro experiments, it was also found that mercury ions can be chelated from kidney slices. 4. This product is safe within the range of clinical dosage. LD50: 400-500mg/Kg for dogs when taken orally; for rats with severe vitamin B1 deficiency, 20mg/Kg was found to be lethal in intraperitoneal injection animal experiments. There is currently insufficient test data to ensure the safety of pregnant women. Clinical side effects are rare. Among the side effects that have been recorded, the main one is skin allergies, and the drug should be stopped at this time.

1. This product is widely used to treat diabetic neuropathy. In vitro tests show that this product can reduce lipid oxidation in nerve tissue, and this product may prevent protein glycosylation: and can inhibit aldose reductase, thereby preventing glucose or galactose from being converted into sorbitol. 2. Animal experiments suggest that lipoic acid can prevent the development of diabetes, promote the utilization of glucose, and prevent neuropathy caused by hyperglycemia. Lipoic acid is a strong antioxidant in both water-soluble and oil-soluble matrices. Both lipoic acid and its reduced form, dilipoic acid, can play an antioxidant role. They can directly or indirectly promote the regeneration of vitamin C and vitamin E in the body. Studies have shown that lipoic acid can increase the levels of glutathione and coenzyme Q10 in cells. 3. Lipoic acid can chelate certain metal ions (such as copper, manganese, and zinc) to form stable chelates. In animal models, it has been shown that it can protect against arsenic poisoning and reduce liver toxicity after chromium poisoning. In in vitro experiments, it was also found that mercury ions can be chelated from kidney slices. 4. This product is safe within the range of clinical dosage. LD50: 400-500mg/Kg for dogs when taken orally; for rats with severe vitamin B1 deficiency, 20mg/Kg was found to be lethal in intraperitoneal injection animal experiments. There is currently insufficient test data to ensure the safety of pregnant women. Clinical side effects are rare. Among the side effects that have been recorded, the main one is skin allergies, and the drug should be stopped at this time.

This product is a second-generation sulfonylurea hypoglycemic drug with a strong effect. Its mechanism is to selectively act on pancreatic islet cells to promote insulin secretion and increase insulin release after eating glucose, so that the production and output of glycogen are inhibited. Animal experiments and clinical use have proven that this product can reduce platelet aggregation and adhesion, and help prevent and treat diabetic microangiopathy.

Not yet clear