The antibacterial spectrum is similar to that of rifampicin. It has good antibacterial activity against tuberculosis and leprosy. When the dosage is 1/3 of that of rifampicin, similar or higher efficacy can be achieved. It has a good effect on Staphylococcus aureus and has certain antibacterial activity against some Escherichia coli. In addition, it also has an inhibitory effect on Chlamydia trachomatis.

Extract from the above information

Antiepileptic drug, antiarrhythmic drug; increases cellular sodium ion outflow, reduces sodium ion inflow, stabilizes nerve cell membrane; shortens action potential interval and effective refractory period, inhibits calcium ion influx, reduces myocardial automaticity; has anti-neuralgia and skeletal muscle relaxation effects; inhibits the synthesis (or) secretion of collagenase by skin fibroblasts; accelerates vitamin D metabolism; has anti-folate effect; has inhibitory effect on the hematopoietic system; can cause allergic reactions; has enzyme induction effect; intravenous administration can dilate peripheral blood vessels.

Antiepileptic drugs, antiarrhythmic drugs. The therapeutic dose does not cause sedation and hypnosis, and has a selective antagonistic effect on the tonic phase of super-strong electric shock and convulsion, but is ineffective or even aggravates the spasm phase. Therefore, it is effective for grand mal epileptic seizures, but ineffective for absence seizures. It increases the outflow of sodium ions from cells, reduces the inflow of sodium ions, stabilizes the nerve cell membrane, increases the excitation threshold, and reduces the spread of high-frequency discharges in the lesion. It shortens the action potential interval and the effective refractory period, inhibits the inflow of calcium ions, reduces myocardial autonomy, inhibits the sympathetic center, has an inhibitory effect on the ectopic rhythm points of the atria and ventricles, and increases the thresholds for atrial fibrillation and ventricular fibrillation.

This product can compete with histamine released from tissues for H2 receptors on effector cells, thereby preventing the onset of allergic reactions and relieving the spasmodic and congestive effects of histamine.