Non-steroidal anti-inflammatory drugs have anti-inflammatory, antipyretic and analgesic effects. The mechanism of action is to reduce the synthesis of prostaglandins by inhibiting cyclooxygenase. It prevents the formation of pain nerve impulses in inflammatory tissues and inhibits inflammatory reactions, including inhibiting the chemotaxis of leukocytes and the release of lysosomal enzymes.

This product is a diamine oxidase inhibitor, which has a more obvious dilating effect on cerebrovascular and cardiovascular systems, especially on the vertebral artery system, significantly increasing the blood flow of the heart, brain and peripheral circulation, improving blood circulation, and lowering systemic blood pressure. In addition, it can increase the blood flow of the cochlea and anterior base, thereby eliminating inner ear vertigo, tinnitus and ear closure, and can also increase capillary permeability, promote the absorption of extracellular fluid, and eliminate lymphatic edema; it can counteract the vasoconstrictive effect of catecholamines and lower arterial pressure, and has the effect of inhibiting plasma coagulation and ADP-induced platelet aggregation, can prolong the time of thrombosis in vitro in rats, and has a slight diuretic effect.

Antibacterial effect