Contains pancreatic lipase, pancreatic amylase, and trypsin, which can break down fat into glycerol and fatty acids, starch into sugar, and protein into peptone

Can convert protein into albumen and peptone

It has the effect of preventing myocardial ischemia-reperfusion injury. It reduces the content of malondialdehyde (MDA) in isolated rat myocardium, reduces tissue calcium accumulation, reduces the content of protein and lactate dehydrogenase (LDH) in coronary artery outflow fluid, and increases the calcium adenosine triphosphatase activity of myocardial sarcoplasmic reticulum.

It has the effect of preventing myocardial ischemia-reperfusion injury. It reduces the content of malondialdehyde (MDA) in isolated rat myocardium, reduces tissue calcium accumulation, reduces the content of protein and lactate dehydrogenase (LDH) in coronary artery outflow fluid, and increases the calcium adenosine triphosphatase activity of myocardial sarcoplasmic reticulum.

It has the effect of preventing myocardial ischemia-reperfusion injury. It reduces the content of malondialdehyde (MDA) in isolated rat myocardium, reduces tissue calcium accumulation, reduces the content of protein and lactate dehydrogenase (LDH) in coronary artery outflow fluid, and increases the calcium adenosine triphosphatase activity of myocardial sarcoplasmic reticulum.

It has the effect of preventing myocardial ischemia-reperfusion injury. It reduces the content of malondialdehyde (MDA) in isolated rat myocardium, reduces tissue calcium accumulation, reduces the content of protein and lactate dehydrogenase (LDH) in coronary artery outflow fluid, and increases the calcium adenosine triphosphatase activity of myocardial sarcoplasmic reticulum.

It has the effect of preventing myocardial ischemia-reperfusion injury. It reduces the content of malondialdehyde (MDA) in isolated rat myocardium, reduces tissue calcium accumulation, reduces the content of protein and lactate dehydrogenase (LDH) in coronary artery outflow fluid, and increases the calcium adenosine triphosphatase activity of myocardial sarcoplasmic reticulum.

It has the effect of preventing myocardial ischemia-reperfusion injury. It reduces the content of malondialdehyde (MDA) in isolated rat myocardium, reduces tissue calcium accumulation, reduces the content of protein and lactate dehydrogenase (LDH) in coronary artery outflow fluid, and increases the calcium adenosine triphosphatase activity of myocardial sarcoplasmic reticulum.

It has the effect of preventing myocardial ischemia-reperfusion injury. It reduces the content of malondialdehyde (MDA) in isolated rat myocardium, reduces tissue calcium accumulation, reduces the content of protein and lactate dehydrogenase (LDH) in coronary artery outflow fluid, and increases the calcium adenosine triphosphatase activity of myocardial sarcoplasmic reticulum.

This product is a coenzyme that improves the body's metabolism and participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotides in the body. It is also the main source of energy in the body. When energy is needed for absorption, secretion, muscle contraction and biochemical synthesis reactions in the body, adenosine triphosphate is decomposed into adenosine diphosphate and phosphate groups, and energy is released at the same time. Animal experiments have shown that this product can inhibit the slow calcium ion influx of slow-reacting fibers, block or delay the forward conduction in the atrioventricular node reentry pathway, and a large dose may also block or delay the forward and reverse conduction of the bypass; in addition, it also has a short-term and strong effect of enhancing the vagus nerve, thereby terminating arrhythmias caused by atrioventricular node reentry and bypass reentry mechanisms.

This product is a coenzyme that improves the body's metabolism and participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotides in the body. It is also the main source of energy in the body. When energy is needed for absorption, secretion, muscle contraction and biochemical synthesis reactions in the body, adenosine triphosphate is decomposed into adenosine diphosphate and phosphate groups, and energy is released at the same time. Animal experiments have shown that this product can inhibit the slow calcium ion influx of slow-reacting fibers, block or delay the forward conduction in the atrioventricular node reentry pathway, and a large dose may also block or delay the forward and reverse conduction of the bypass; in addition, it also has a short-term and strong effect of enhancing the vagus nerve, thereby terminating arrhythmias caused by atrioventricular node reentry and bypass reentry mechanisms.