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Founded in:
2000-12-21 -
Country:
China -
Address:
No. 6 and No. 8, Section 2, Wangjiang Road, Yutang Town, Longmatan District, Luzhou City -
Tax NO.:
91510500723205632X -
Registered Funds:
36.1 million yuan -
Website:
-
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| (±)5-[[4-[2-(5-ethyl-2-pyridyl)ethoxy]phenyl]methyl]-2,4-thiazolidinedione hydrochloride |
This product belongs to the class of thiazolidinediones oral antidiabetic drugs, and is a highly selective peroxisome proliferator-activated receptor γ (PPARγ) agonist, which controls blood sugar levels by increasing peripheral and liver insulin sensitivity. Its main mechanism of action is to activate PPARγ nuclear receptors in tissues such as fat, skeletal muscle and liver where insulin acts, thereby regulating the transcription of insulin-responsive genes and controlling the production, transport and utilization of blood sugar.
More
This product belongs to the class of thiazolidinediones oral antidiabetic drugs, and is a highly selective peroxisome proliferator-activated receptor γ (PPARγ) agonist, which controls blood sugar levels by increasing peripheral and liver insulin sensitivity. Its main mechanism of action is to activate PPARγ nuclear receptors in tissues such as fat, skeletal muscle and liver where insulin acts, thereby regulating the transcription of insulin-responsive genes and controlling the production, transport and utilization of blood sugar. |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| (±)5-[[4-[2-(5-ethyl-2-pyridyl)ethoxy]phenyl]methyl]-2,4-thiazolidinedione hydrochloride |
This product belongs to the class of thiazolidinediones oral antidiabetic drugs, and is a highly selective peroxisome proliferator-activated receptor γ (PPARγ) agonist, which controls blood sugar levels by increasing peripheral and liver insulin sensitivity. Its main mechanism of action is to activate PPARγ nuclear receptors in tissues such as fat, skeletal muscle and liver where insulin acts, thereby regulating the transcription of insulin-responsive genes and controlling the production, transport and utilization of blood sugar.
More
This product belongs to the class of thiazolidinediones oral antidiabetic drugs, and is a highly selective peroxisome proliferator-activated receptor γ (PPARγ) agonist, which controls blood sugar levels by increasing peripheral and liver insulin sensitivity. Its main mechanism of action is to activate PPARγ nuclear receptors in tissues such as fat, skeletal muscle and liver where insulin acts, thereby regulating the transcription of insulin-responsive genes and controlling the production, transport and utilization of blood sugar. |
0 | ||
| (±) 5-[[4-[2-(5-ethyl-2-pyridyl)ethoxy]phenyl]methyl]-2,4-thiazolidinedione hydrochloride |
This product belongs to the class of thiazolidinediones oral antidiabetic drugs, and is a highly selective peroxisome proliferator-activated receptor γ (PPARγ) agonist, which controls blood sugar levels by increasing peripheral and liver insulin sensitivity. Its main mechanism of action is to activate PPARγ nuclear receptors in tissues such as fat, skeletal muscle and liver where insulin acts, thereby regulating the transcription of insulin-responsive genes and controlling the production, transport and utilization of blood sugar.
More
This product belongs to the class of thiazolidinediones oral antidiabetic drugs, and is a highly selective peroxisome proliferator-activated receptor γ (PPARγ) agonist, which controls blood sugar levels by increasing peripheral and liver insulin sensitivity. Its main mechanism of action is to activate PPARγ nuclear receptors in tissues such as fat, skeletal muscle and liver where insulin acts, thereby regulating the transcription of insulin-responsive genes and controlling the production, transport and utilization of blood sugar. |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| D-Glucosamine hydrochloride |
It stimulates chondrocytes to produce proteoglycans with normal polymer structures, improves the repair ability of chondrocytes, inhibits enzymes that damage cartilage such as collagenase and phospholipase A2, and prevents the production of superoxide free radicals that damage cells, promotes the repair and reconstruction of cartilage matrix, delays the pathological process of bone and joint pain and the progression of the disease, improves joint movement, and relieves pain.
More
It stimulates chondrocytes to produce proteoglycans with normal polymer structures, improves the repair ability of chondrocytes, inhibits enzymes that damage cartilage such as collagenase and phospholipase A2, and prevents the production of superoxide free radicals that damage cells, promotes the repair and reconstruction of cartilage matrix, delays the pathological process of bone and joint pain and the progression of the disease, improves joint movement, and relieves pain. |
0.75g | 66-84-2 | 17 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| PAEONIAE RADIX ALBA |
|
0 | ||
| Motherwort Herb P.E. |
|
0 | ||
| Angelicae Sinensis Radix |
|
0 | ||
| CYPERI RHIZOMA |
|
0 | ||
| NOTOGINSENG RADIX ET RHIZOMA |
|
0 | ||
| Codonopsis |
|
0 | ||
| Ophiopogonis Radix |
|
0 | ||
| Mugwort (charcoal) |
|
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Bupleurum |
|
0 | ||
| Paeoniae Rubra |
|
0 | ||
| AURANTII FRUCTUS |
|
0 | ||
| Aucklandiae Radix |
|
0 | ||
| Salvia Root P.E Tanshinone IIA 20% |
|
0 | ||
| Yanhusuo |
|
0 | ||
| Zedoary turmeric |
|
0 | ||
| COPTIDISRHIZOMA |
|
0 | ||
| EUODIAE FRUCTUS |
|
0 | ||
| Chrysophanic acid |
|
481-74-3 | 0 | |
| Codonopsis |
|
0 | ||
| DGL |
|
0 |