1. Anti-tumor effect: It shows high anti-tumor activity against mouse leukemia L-1210, and has a wide anti-tumor spectrum against mouse transplanted tumors, showing excellent effects on lymphocytic leukemia L1210, granulocytic leukemia C-1498, plasmacytoma X-5563 (ascites type), Ehrlich's ascites carcinoma, and dural sarcoma MS-147; this agent has a life-extending effect on mice with lymphocytic leukemia L1210 and intracerebral transplantation of malignant glioma cells induced by methylcholanthrene. 2. Mechanism of action: The main mechanism of action is to alkylate DNA in cells to reduce the molecular weight of DNA and inhibit DNA synthesis.

It strengthens the function of the brainstem reticular formation, especially the ascending reticular formation excitatory system which is closely related to consciousness; strengthens the function of the vertebral system; improves motor paralysis; improves cerebral circulation, promotes brain metabolism by reducing cerebral blood flow resistance, increasing cerebral blood flow, and has a certain effect on promoting brain function recovery and awakening.

Strengthen the function of the brainstem reticular formation, especially the ascending reticular formation excitatory system which is closely related to consciousness; strengthen the function of the vertebral system and improve motor paralysis; improve cerebral circulation, promote brain metabolism by reducing cerebral blood flow resistance and increasing cerebral blood flow, and play a certain role in promoting brain function recovery and awakening.

Extract from the above information

In vitro tests have destructive or inhibitory effects on the morphology or proliferation of cell lines such as liver cancer, esophageal cancer, laryngeal cancer, lung cancer, and cervical cancer; in vivo tests have a certain inhibitory effect on Ehrlich ascites carcinoma, sarcoma-180 and solid liver cancer in mice, and can increase the respiratory control rate and lysosomal enzyme activity of mitochondria in ascites liver cancer H22 mice, interfere with cancer cell division, block it in the M phase, and affect its cycle speed; destroy the cancer cell skeleton (microfilaments, microtubules), affect the ultrastructure of cancer cells, and cause damage to mitochondria, microvilli and plasma membranes; can inhibit cancer cell DNA synthesis, but has no inhibitory effect on normal bone marrow cells, and can increase white blood cells.